Abstract
The current study deals with the stochastic reaction–diffusion epidemic model numerically with two proposed schemes. Such models have many applications in the disease dynamics of wildlife, human life, and others. During the last decade, it is observed that the epidemic models cannot predict the accurate behavior of infectious diseases. The empirical data gained about the spread of the disease shows non-deterministic behavior. It is a strong challenge for researchers to consider stochastic epidemic models. The effect of the stochastic process is analyzed. So, the SIR epidemic model is considered under the influence of the stochastic process. The time noise term is taken as the stochastic source. The coefficient of the stochastic term is a Borel function, and it is used to control the random behavior in the solutions. The proposed stochastic backward Euler scheme and the proposed stochastic implicit finite difference scheme (IFDS) are used for the numerical solution of the underlying model. Both schemes are consistent in the mean square sense. The stability of the schemes is proven with Von-Neumann criteria and schemes are unconditionally stable. The proposed stochastic backward Euler scheme converges toward a disease-free equilibrium and does not converge toward an endemic equilibrium but also possesses negative behavior. The proposed stochastic IFD scheme converges toward disease-free equilibrium and endemic equilibrium. This scheme also preserves positivity. The graphical behavior of the stochastic SIR model is much similar to the classical SIR epidemic model when noise strength approaches zero. The three-dimensional plots of the susceptible and infected individuals are drawn for two cases of endemic equilibrium and disease-free equilibriums. The efficacy of the proposed scheme is shown in the graphical behavior of the test problem for the various values of the parameters.
Similar content being viewed by others
Avoid common mistakes on your manuscript.
1 Introduction
The reaction–diffusion models are used in ecology, and virology to explain various biological phenomena. Such models describe the population or concentration distributed in space under the effect of two phenomena. In population models or concentration models, the diffusion term explains the random motion of the individuals, and their reproduction is described by the reaction term. In epidemiology, reaction–diffusion models are frequently used to understand the phenomena of various diseases such as how the disease starts, how it is affected by the movement of different species, how it can be controlled, and how much time is required to eradicate it from the species. Some researchers worked on the numerical solutions of the epidemic models. A mathematical model was proposed by Wang and Wang [1] for the simulation of the hepatitis B virus infection and introduced a random motion of the virus in the deterministic model proposed by Nowak et al. [2], and they assumed that concentration gradient is proportional to the population flux of the virus. Suryanto et al. proposed a nonstandard FDS for the numerical approximation of the SIR epidemic model having a saturated incidence rate. The results obtained with the given scheme are dynamically consistent with the continuous model [3]. Naik et al. considered the SIR epidemic model to have a Crowley-Martin-type functional response and Holling type-II treatment rate. They used the Homotopy analysis method is used for the analytical solutions of the given model. The authors discussed the stability of the model and conclude that it has two equilibria, one is disease free and the other is endemic [4].
Nauman et al. [5] considered the following epidemic model having nonlinear incidence rate as follows:
with initial condition
and homogeneous Neumann boundary conditions(B.Cs)
Where S(x, t), I(x, t), and R(x, t) represent the susceptible, infected, and recovered individuals, respectively, at any space variable x and time t. The parameters \(\Lambda , \eta , \delta , \sigma \) represent the recruitment rate, natural death rate, the death rate due to disease, and recovery rate of the infected individuals, respectively. The \(d_a, a=1,2\) are the diffusion coefficients, \(\beta \) is the infection parameter, \(c_a, a=1,2,3\) are the arbitrary positive constants, \( \frac{\beta SI}{1+c_1 S+ c_2 I+c_3SI}\) is the nonlinear modified incidence rate. The nonlinear modified incidence rate becomes bilinear if \(c_a=0, a=1,2,3\), and the saturated incidence if \(c_a=0, a=1,2\) or \( a=2,3\). The stability analysis of the above model has been carried out, and it has two equilibria, one is the disease-free equilibrium is \((\lambda /\eta ,0)\) and the other is endemic equilibrium \((S^*, I^*)\) and \(S^*=\frac{2(e'+c_2\lambda )}{\beta -c_1e'+c_2 \eta -c_3\lambda + \sqrt{d'}}\)
\(I^*=\frac{\lambda -\eta S^*}{e'}\) where \(e'=\eta + \delta + \alpha \), \(d'=(\beta - c_1 e'+c_2 \eta -c_3 \lambda )^2 + 4c_3\eta (e'+c_2 \lambda )\). The reproduction number is given as \( R_0=\frac{\beta \lambda }{(\eta +c_1\lambda )(\eta +\delta + \alpha )}\). As Eqs. (1) and (2) are independent of the state variable R, it is quite natural to consider the system of Eqs. (1,2) instead of (1,2,3) because R does not affect the susceptible and infected individuals.
Modeling of physical phenomena is an interesting work. For this purpose, partial differential equations (PDEs) are used to describe the physical behavior [6,7,8,9]. The solution of the PDEs is an active area of research, and various techniques are applied for the solutions of PDEs [10,11,12,13,14,15,16]. Forty years ago, it was a faith that medical science has made tremendous progress in reducing the mortality rate of humans because it is due to improvements in nutrition, drugs, and vaccines. Even then, infectious diseases have been a severe threat to the human and livestock population. The classical epidemic models fail to describe the true behavior of the diseases. So, it is the need of the hour to consider the epidemic models under some stochastic process. So, new literature is required for cases. various researchers are working on the dynamics of stochastic partial differential equations. The authors used the finite difference scheme to analyze the different aspects of various physical phenomena [17,18,19]. Macías-Díaz et al. analyzed the stochastic epidemiological model with a nonstandard finite difference scheme [20]. Raza et al. developed a nonstandard finite difference for the study of the stochastic smoking model and discussed various dynamics [21]. Nauman et al. worked on the numerical approximation of the stochastic fractional epidemic model [22]. Raza et al. found the solution of the stochastic dengue epidemic model [23]. Alkhazzan et al. analyzed the SVIR epidemic model and discussed its various dynamics [24].
The main contributions of this manuscript are as given below:
- (i):
-
The epidemic models cannot predict the accurate behavior of the infectious disease. So, stochastic epidemic models are preferred. We are considering the stochastic epidemics model.
- (ii):
-
Numerical approximation of the underlying model is carried out by two schemes.
- (iii):
-
The stability and consistency of the schemes are analyzed in the mean square sense.
- (iv):
-
The given model has two equilibrium points: one is disease free and the other is endemic equilibrium.
- (v):
-
The numerical simulations of the test problem are drawn for various choices of the parameters.
- (vi):
-
The MATLAB 2015a is used for the graphical behavior of the test problem.
In structure of article, Sect. 2 contains the stochastic epidemic model. The Sect. 3 deals with the two numerical schemes for the given model. The analysis of both schemes is carried out. Section 4 deals with the test problem, and its graphical behavior and discussion are carried out for endemic equilibrium and disease-free equilibrium as well. The last Sect. 5 related to the conclusion of this article.
2 Stochastic epidemic model
During the last decade, it is observed that the epidemic models cannot predict the accurate behavior of the infectious disease. The empirical data gained about the spread of the disease show the non-deterministic behavior [25]. So, various stochastic models are proposed to describe the propagation of the disease in human life such as stochastic semi-mechanistic models [26], the stochastic SIS epidemic models with nonlinear incidence rate [27], the stochastic SIRS epidemic models having saturated incidence rate [28], and the SIS nonlinear stochastic epidemiological models having incidence rate and double epidemic hypothesis[29]. It is quite natural to consider the epidemic under the effect of some perturbation.
We are taking the stochastic version of the system of the Eqs. (1,2) under the effect of time noise as follows:
with initial conditions
and homogeneous Neumann boundary conditions(B.Cs)
Where \(\alpha _a, a=1,2\) are the noise strength of the time noise, \(\dot{B}_a(t), a=1,2\) is the time noise and we consider the standard Wiener process for stochastic behavior of the epidemic model. Various researchers are working on the numerical study of SDEs. Cai et al. worked on the solution of the stochastic epidemic model analytically and numerically [30]. Meng et al. proposed a stochastic eco-epidemiological model with time delay and a general incidence rate. They discussed the unique existence of a positive global solution with positive initial conditions. They also established the stochastic analysis of the model [31]. The authors considered a stochastic hepatitis B model and derived the different conditions for the extinction and persistence of the disease [32]. In [33], the authors used the numerical technique for the study of the \(Covid-19\) model. The authors worked on the numerical approximations of the stochastic partial differential equations [34]. Zho et al. worked on the stochastic SIR model and discussed the different dynamics of the model. They show that the model has a unique global positive solution and the solution oscillates around the equilibrium points under some conditions [35]. Some more work on the numerical solutions of the differential equations can be seen [36,37,38,39,40,41]. We are using two numerical schemes namely, one is the proposed stochastic backward Euler scheme and the other is the proposed stochastic implicit FDS. The analysis of the schemes and after that numerical simulations will be carried out for the efficacy of our findings.
3 Numerical schemes
We have divided the \([0,L]\times [0,T]\) into \(N\times M\) grid points, and the stepsizes are \(l=T/M\), \(m=L/N\), respectively. The grid points are \(x_a=am, \qquad a=0,1,2,3,4,\ldots , N\),
\(t_b=bl, \qquad b=0,1,2,3,4,\ldots , M\).
The proposed stochastic backward Euler scheme for Eqs. (8,9) is
The proposed stochastic implicit finite difference scheme for Eqs. (8,9) is
where \(R_1=l d_1/m^2\), and \(R_2=l d_2/m^2\). The \(S_a^b, I_a^b\) are the numerical approximations of the S, I at the point (am, bl).
3.1 Consistency of a scheme
Definition
[42,43,44]. In the mean square sense, a SFDS \(A|_{a}^{b}U|_{a}^{b}= F|_{a}^{b}\) is consistent with stochastic PDE \( AU=F \) at a point (x, t), if there exist any continuously differentiable function \(\Xi =\Xi (x,t)\), then
as \( m\rightarrow 0, l \rightarrow 0 \) and \((a m, (b+1)l)\rightarrow (x, t)\).
3.2 Von-Neumann analysis
In this technique, \(S|_{a}^{b}\) is taken as follow,
and \(\hat{S|}^b_a\) is defined as
here, \(\psi \) is variable, and by replacing values in a given PDE,
Now, the necessary and sufficient condition of this method is given below [45],
where, \(\chi \) is a constant.
3.3 Consistency of the proposed stochastic backward Euler scheme
The consistency analysis is carried out in the mean square sense.
Theorem 1
In a mean square sense, the stochastic FD scheme in Eqs. (14,15) for the variables S, I is consistent with Eqs. (8, 9).
Proof
By assumption, S(x, t) and I(x, t) are smooth functions and by using \( A(f) = \int ^{(b+1)l}_{bl} {f} \textrm{d}r \) on Eq. (8). We get
By using the proposed scheme on Eq. (8)
The above two equations can be expressed as in the mean square
Due to the Itô integrals’ square feature, we can
\(E|A(S)|_{a}^{b}-A|_{a}^{b} S|^2\rightarrow 0 \) as \( a\rightarrow \infty , b\rightarrow \infty \)., The scheme for S is consistent (8). Now, the consistency of the (9) is given below
By using scheme on Eq. (8),
The above two equations can be expressed as in the mean square
Due to the Itô integrals’ square feature, we can
\(E|A(I)|_{a}^{b}-A|_{a}^{b} I|^2\rightarrow 0 \) as \( a\rightarrow \infty , b\rightarrow \infty \). The scheme for I is consistent with (9).
3.4 Consistency of proposed stochastic implicit finite difference scheme
The consistency analysis is carried out in the mean square sense.
Theorem 2
In a mean square sense, the stochastic FD scheme in Eqs. (16, 16) for the variables S, I is consistent with Eqs. (8,9).
Proof
By assumption S(x, t) and I(x, t) are smooth functions and by using the \( A(f) = \int ^{(b+1)l}_{bl} {f} \textrm{d}r \) on Eq. (8). We get
By using the proposed scheme- on Eq. (8),
The above two equations can be expressed as in the mean square
Due to the Itô integrals’ square feature, we can
\(E|A(S)|_{a}^{b}-A|_{a}^{b} S|^2\rightarrow 0 \) as \( a\rightarrow \infty , b\rightarrow \infty , \) so this proposed scheme for S is consistent with stochastic PDE (8).
Now, the consistency of the (9) is given as
By using the proposed scheme on Eq. (9)
The above two equations can be expressed as in the mean square
Due to the Itô integrals’ square feature, we can
\(E|A(I)|_{a}^{b}-A|_{a}^{b} I|^2\rightarrow 0 \) as \( a\rightarrow \infty , b\rightarrow \infty \). The scheme for I is consistent with Eq. (9).
3.5 Stability of proposed stochastic backward Euler scheme
The linear stability analysis is carried out.
Theorem 3
The scheme for S, I by Eqs. (14,15) is unconditionally stable.
Proof
We are using the Von-Neumann technique for the stability analysis. So, by linearizing Eq. (14) and keeping the coefficient term frozen, we have
By replacing the value of S from Eq. (19)
Using the Wiener process’ independent state, the amplification factor can now be expressed as
As always
and let \(\bigg |\frac{\alpha _1}{1+4R_1 \sin ^2(\frac{m \psi }{2})} \bigg |^2= \chi \), so,
So, scheme for S is stable.
Now, to discuss the stability of the scheme for I, by linearizing Eq. (9) and keeping the coefficient term frozen, we have
By replacing the value of I from Eq. (19)
Using the Wiener process’ independent state, the amplification factor can now be expressed as
As always
and let \(\bigg |\frac{\alpha _2}{1+4R_2 \sin ^2(\frac{m \psi }{2})} \bigg |^2= \chi \), so
So, scheme for I is stable.
3.6 Stability analysis of proposed stochastic implicit scheme
The linear stability analysis is carried out.
Theorem 4
The scheme for S, I by Eqs. (16,16) is unconditionally stable.
Proof
We are using the Von-Neumann technique for the stability analysis. So, by linearizing Eq. (14) and keeping the coefficient term frozen, we have
By replacing the value of S from Eq. (19),
Using the Wiener process’ independent state, the amplification factor can now be expressed as
As always
and let \(\bigg |\frac{\alpha _1}{1+\eta l+4R_1 \sin ^2(\frac{m \psi }{2})} \bigg |^2= \chi \), so
So, proposed for S is stable.
Now, to discuss the stability of the scheme for I, by linearizing Eq. (16) and keeping the coefficient term frozen, we have
By replacing the value of I from Eq. (19)
Using the Wiener process’ independent state, the amplification factor can now be expressed as
As always
and let \(\bigg |\frac{\alpha _2}{1+l(\eta +\delta +\sigma )+4R_2 \sin ^2(\frac{m \psi }{2})} \bigg |^2= \chi \), so
So, the scheme for I is stable.
4 Numerical results
with initial conditions as
and having homogeneous Neumann boundary conditions.
4.1 Disease-free equilibrium of test problem
All the figures of disease-free equilibrium are plotted for different values of the parameters given in Table 1. For the given values of the parameters, the reproductive number for the disease-free equilibrium is \(R_0=0.9286 <1\) with \(\beta =0.2\). For the system of the Eqs. (8,9), the disease-free equilibrium \(DFE=(\lambda /\eta ,0)\) has the value (5,0) for \(\beta =0.2\). Figures 1, 2 are plotted for noise strength equal to zero, i.e., without stochastic behavior. Figure 1 is acquired by using the proposed stochastic backward Euler scheme and the susceptible and infected individuals converge toward the disease-free equilibrium, and the solution has positive values. Figure 2 is acquired by using the proposed stochastic implicit FDS. The susceptible and infected individuals converge toward disease-free equilibrium and the solution preserves the positivity and boundedness. Positivity and boundedness are the important dynamics of the model. Figures 3, 4 are drawn for the noise strength have to value 0.5, and stochastic behavior is observed. Figure 3 is acquired by the proposed stochastic Backward Euler scheme, and it converges toward DFE and the solutions also preserve the positivity. Figure 4 is acquired by the proposed stochastic IFD scheme and the susceptible and infected individuals converge toward DFE. So, both schemes converge toward DFE with and without stochastic phenomena and preserve positivity.
4.2 Endemic equilibrium \((\beta = 5, )\) of the problem
All the figures of endemic equilibrium are plotted for different values of the parameters given in Table 1. For the given values of the parameters, the reproductive number for the endemic equilibrium is \(R_0=35.2143\) with \(\beta =5\). For the system of the Eqs. (8,9), the endemic equilibrium \(EE=(S^*,I^*)\) has the value (0.1440,0.6937) for \(\beta =5\). Figures 5, 6 are drawn for noise strength having value zero, i.e., without stochastic behavior. Figure 5 is drawn by the proposed stochastic backward Euler scheme. The susceptible and infected individuals do not converge toward the endemic equilibrium. The solutions possess negative behavior, and it is meaningless in the disease dynamics. Figure 6 is plotted by the proposed stochastic IFD scheme and the susceptible and infected individuals converge toward \((S^*, I^*)\). The solutions preserve positivity and boundedness as well. Figures 7, 8 are drawn for the noise strength having a value equal to 0.5. Figure 7 is drawn by the proposed stochastic backward Euler scheme. The susceptible and infected individuals do not converge toward the endemic equilibrium and also possess negative behavior and it is meaningless. Figure 8 is plotted by the proposed stochastic IFD scheme and the susceptible and infected individuals converge toward \((S^*, I^*)\). The solutions preserve positivity. So, the proposed stochastic backward Euler scheme does not have the required results because the given scheme has negative solutions with and without stochastic behavior. But the proposed stochastic IFD scheme has convergent behavior as well preserves positivity.
4.3 Graphical behavior
The plots are drawn for the efficacy of our findings.
5 Conclusion
The stochastic reaction–diffusion epidemic model has been numerically investigated by two time-efficient novel schemes. The proposed stochastic backward Euler scheme is consistent with the given system of equations. It is also unconditionally stable, and its stability analysis is carried out by using the von-Neumann criteria. The suggested stochastic IFD approach is compatible with the provided equation system. Additionally, it is unconditionally stable, and the von Neumann criteria are used to analyze its stability. The system has two equilibriums: an endemic equilibrium and a disease-free equilibrium. For the disease-free equilibrium, both schemes converge toward the equilibrium and preserve positivity. For the endemic equilibrium, the proposed stochastic backward Euler scheme did not show the convergent behavior toward the endemic equilibrium and it also shows the negative behavior of the solution and which is meaningless in the population dynamics. On the other hand, the proposed stochastic IFD scheme shows the convergent behavior toward the endemic equilibrium and also preserves positivity. The graphical behavior of the test problems shows that the proposed stochastic IFD scheme has an advantage over the proposed stochastic backward Euler scheme. The graphical results for the test problem are drawn that show the efficacy of our proposed stochastic IFD scheme.
Data availability
Data sharing not applicable to this article.
References
K. Wang, W. Wang, Propagation of HBV with spatial dependence. Math. Biosci. 210(1), 78–95 (2007)
M.A. Nowak, S. Bonhoeffer, A.M. Hill, R. Boehme, H.C. Thomas, H. McDade, Viral dynamics in hepatitis B virus infection. Proc. Natl. Acad. Sci. 93(9), 4398–4402 (1996)
A. Suryanto, I. Darti, On the nonstandard numerical discretization of SIR epidemic model with a saturated incidence rate and vaccination. AIMS Math. 6, 141–155 (2021)
P.A. Naik, J. Zu, M. Ghoreishi, Stability analysis and approximate solution of SIR epidemic model with Crowley–Martin type functional response and holling type-II treatment rate by using homotopy analysis method. J. Appl. Anal. Comput. 10(4), 1482–1515 (2020)
N. Ahmed, A. Korkamaz, M.A. Rehman, M. Rafiq, M. Ali, M.O. Ahmad, Computational modelling and bifurcation analysis of reaction diffusion epidemic system with modified nonlinear incidence rate. Int. J. Comput. Math. 98(3), 517–535 (2021)
I. Ahmad, M.N. Khan, M. Inc, H. Ahmad, K.S. Nisar, Numerical simulation of simulate an anomalous solute transport model via local meshless method. Alex. Eng. J. 59(4), 2827–2838 (2020)
H. Ahmad, A. Akgül, T.A., Khan, P.S. Stanimirovic, Y.M. Chu, New perspective on the conventional solutions of the nonlinear time-fractional partial differential equations. Complexity 2020, 1–10 (2020)
H. Ahmad, T.A. Khan, P.S. Stanimirovic, I. Ahmad, Modified variational iteration technique for the numerical? solution of fifth order KdV-type equations. J. Appl. Comput. Mech. 6, 1220–1227 (2020)
H. Ahmad, A.R. Seadawy, T.A. Khana, modified variational iteration algorithm to find approximate solutions of nonlinear Parabolic equation. Math. Comput. Simul. 177, 13–23 (2020)
I. Ahmad, H. Ahmad, M. Inc, S.W. Yao, B. Almohsen, Application of local meshless method for the solution of two term time fractional-order multi-dimensional PDE arising in heat and mass transfer. Therm. Sci. 24(Suppl. 1), 95–105 (2020)
M. Inc, M.N. Khan, I. Ahmad, S.W. Yao, H. Ahmad, P. Thounthong, Analysing time-fractional exotic options via efficient local meshless method. Results Phys. 19, 103385 (2020)
M. Nawaz Khan, I. Ahmad, H. Ahmad, A Radial Basis Function Collocation Method for Space-dependent? Inverse Heat Problems. J. Appl. Comput. Mech. (2020)
N.A. Shah, I. Ahmad, O. Bazighifan, A.E. Abouelregal, H. Ahmad, Multistage optimal homotopy asymptotic method for the nonlinear Riccati ordinary differential equation in nonlinear physics. Appl. Math. 14(6), 1009–1016 (2020)
F. Wang, S.N. Ali, I. Ahmad, H. Ahmad, K.M. Alam, P. Thounthong, Solution of Burgers’ equation appears in fluid mechanics by multistage optimal homotopy asymptotic method. Therm. Sci. 26(1 Part B), 815–821 (2022)
X. Liu, M. Ahsan, M. Ahmad, M. Nisar, X. Liu, I. Ahmad, H. Ahmad, Applications of Haar wavelet-finite difference hybrid method and its convergence for hyperbolic nonlinear Schrö dinger equation with energy and mass conversion. Energies 14(23), 7831 (2021)
M. Ahsan, S. Lin, M. Ahmad, M. Nisar, I. Ahmad, H. Ahmed, X. Liu, A Haar wavelet-based scheme for finding the control parameter in nonlinear inverse heat conduction equation. Open Phys. 19(1), 722–734 (2021)
M.W. Yasin, N. Ahmed, M.S. Iqbal, M. Rafiq, A. Raza, Akgül, A., Reliable numerical analysis for stochastic reaction–diffusion system. Physica Scripta 98(1), 015209 (2022)
X. Wang, M.W. Yasin, N. Ahmed, M. Rafiq, M. Abbas, Numerical approximations of stochastic Gray–Scott model with two novel schemes. AIMS Math. 8(3), 5124–5147 (2023)
M.W. Yasin, N. Ahmed, M.S. Iqbal, A. Raza, M. Rafiq, E.M.T. Eldin, I. Khan, Spatio-temporal numerical modeling of stochastic predator–prey model. Sci. Rep. 13(1), 1990 (2023)
J.E. Macías-Díaz, A. Raza, N. Ahmed, M. Rafiq, Analysis of a nonstandard computer method to simulate a nonlinear stochastic epidemiological model of coronavirus-like diseases. Comput. Methods Prog. Biomed. 204, 106054 (2021)
A. Raza, M. Rafiq, N. Ahmed, I. Khan, K.S. Nisar, Z. Iqbal, A structure preserving numerical method for solution of stochastic epidemic model of smoking dynamics. Comput. Mater. Continua 65(01), 263–278 (2020)
N. Ahmed, J.E., Macías-Díaz, A. Raza, D. Baleanu, M. Rafiq, Z. Iqbal, M.O. Ahmad, Design analysis and comparison of a nonstandard computational method for the solution of a general stochastic fractional epidemic model. Axioms 11(1), 10 (2021)
A. Raza, M.S. Arif, M. Rafiq, A reliable numerical analysis for stochastic dengue epidemic model with incubation period of virus. Adv. Differ. Equ. 2019(1), 1–19 (2019)
A. Alkhazzan, J. Wang, Y. Nie, K. Hattaf, A new stochastic split-step \(\theta \)-nonstandard finite difference method for the developed SVIR epidemic model with temporary immunities and general incidence rates. Vaccines 10(10), 1682 (2022)
K. Chatterjee, K. Chatterjee, A. Kumar, S. Shankar, Healthcare impact of COVID-19 epidemic in India: a stochastic mathematical model. Med. J. Armed Forces India 76(2), 147–155 (2020)
S. Funk, A. Camacho, A.J. Kucharski, R.M. Eggo, W.J. Edmunds, Real-time forecasting of infectious disease dynamics with a stochastic semi-mechanistic model. Epidemics 22, 56–61 (2018)
N. Gao, Y. Song, X. Wang, J. Liu, Dynamics of a stochastic SIS epidemic model with nonlinear incidence rates. Adv. Differ. Equ. 2019(1), 1–19 (2019)
Y. Song, A. Miao, T. Zhang, X. Wang, J. Liu, Extinction and persistence of a stochastic SIRS epidemic model with saturated incidence rate and transfer from infectious to susceptible. Adv. Differ. Equ. 2018(1), 1–11 (2018)
A. Miao, X. Wang, T. Zhang, W. Wang, B.G. Sampath Aruna Pradeep, Dynamical analysis of a stochastic SIS epidemic model with nonlinear incidence rate and double epidemic hypothesis. Adv. Differ. Equ. 2017(1), 1–27 (2017)
Y. Cai, J. Jiao, Z. Gui, Y. Liu, W. Wang, Environmental variability in a stochastic epidemic model. Appl. Math. Comput. 329, 210–226 (2018)
X. Meng, F. Li, S. Gao, Global analysis and numerical simulations of a novel stochastic eco-epidemiological model with time delay. Appl. Math. Comput. 339, 701–726 (2018)
T. Khan, A. Khan, G. Zaman, The extinction and persistence of the stochastic hepatitis B epidemic model. Chaos Solitons Fractals 108, 123–128 (2018)
X. Chen, J. Li, C. Xiao, P. Yang, Numerical solution and parameter estimation for uncertain SIR model with application to COVID-19. Fuzzy Optim. Decis. Mak. 20(2), 189–208 (2021)
M.Z. Baber, A.R. Seadway, M.S. Iqbal, N. Ahmed, M.W. Yasin, M.O. Ahmed, Comparative analysis of numerical and newly constructed soliton solutions of stochastic Fisher-type equations in a sufficiently long habitat. Int. J. Mod. Phys. B 2350155 (2022)
M. Zhao, H. Zhao, Asymptotic behavior of global positive solution to a stochastic SIR model incorporating media coverage. Adv. Differ. Equ. 2016(1), 1–17 (2016)
M.W. Yasin, M.S. Iqbal, N. Ahmed, Akgül, A., Raza, A., Rafiq, M., & Riaz, M. B., Numerical scheme and stability analysis of stochastic Fitzhugh–Nagumo model. Results Phys. 32, 105023 (2022)
M.W. Yasin, M.S. Iqbal, A.R. Seadawy, M.Z. Baber, M. Younis, S.T. Rizvi, Numerical scheme and analytical solutions to the stochastic nonlinear advection diffusion dynamical model. Int. J. Nonlinear Sci. Numer. Simul. (2021)
M.S. Iqbal, M.W. Yasin, N. Ahmed, A. Akgül, M. Rafiq, A. Raza, Numerical simulations of nonlinear stochastic Newell–Whitehead–Segel equation and its measurable properties. J. Comput. Appl. Math. 418, 114618 (2023)
D. Uma, H. Jafari, S. Raja Balachandar, S.G. Venkatesh, An approximation method for stochastic heat equation driven by white noise. Int. J. Appl. Comput. Math. 8(6), 1–18 (2022)
M. Kamrani, S.M. Hosseini, The role of coefficients of a general SPDE on the stability and convergence of a finite difference method. J. Comput. Appl. Math. 234(5), 1426–1434 (2010)
Z. Zhang, G.E. Karniadakis, Numerical methods for stochastic partial differential equations with white noise (Springer International Publishing, Heidelberg/Berlin, Germany, 2017)
B. Gustafsson, On the convergence rate for difference approximations to mixed initial boundary value problems (Uppsala University, Department of Computer Sciences, 1971)
J. Gary, A generalization of the Lax–Richtmyer theorem on finite difference schemes. SIAM J. Numer. Anal. 3(3), 467–473 (1966)
R.D. Richtmyer, K.W. Morton, Difference methods for initial-value problems. Malabar (1994)
C. Roth, Approximations of solutions of a first order stochastic partial differential equation. Report, Institut Optimierung und Stochastik, Martin-Luther-Universitat Halle-Wittenberg, Halle (1989)
Author information
Authors and Affiliations
Corresponding author
Ethics declarations
Competing interests
The authors declare no competing interest.
Rights and permissions
Springer Nature or its licensor (e.g. a society or other partner) holds exclusive rights to this article under a publishing agreement with the author(s) or other rightsholder(s); author self-archiving of the accepted manuscript version of this article is solely governed by the terms of such publishing agreement and applicable law.
About this article
Cite this article
Ahmed, N., Yasin, M.W., Iqbal, M.S. et al. A dynamical study on stochastic reaction diffusion epidemic model with nonlinear incidence rate. Eur. Phys. J. Plus 138, 350 (2023). https://doi.org/10.1140/epjp/s13360-023-03936-z
Received:
Accepted:
Published:
DOI: https://doi.org/10.1140/epjp/s13360-023-03936-z