Abstract
Hypoperfusion could be defined as “a syndrome leading to widespread cellular hypoxia and vital organ dysfunction” [1], or “an inappropriate balance of substrate supply and demand at a cellular level” [2]; from a clinical point of view, whatever the cause, it is “a state in which reduction of effective tissue perfusion leads first to reversible, and then to irreversible cellular injury” [3]. Perhaps, after some intriguing insights about the subject, we should also include among the hypoperfusion syndromes any pathological O2 utilization at the cellular level, the so-called “dysoxia” [4, 5].
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Schiraldi, F., Derosa, A.R., Paladino, F. (1998). Metabolic Disorders and Electrolyte Derangement during Hypoperfusion Syndrome. In: Gullo, A. (eds) Anaesthesia, Pain, Intensive Care and Emergency Medicine - A.P.I.C.E.. Springer, Milano. https://doi.org/10.1007/978-88-470-2278-2_75
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DOI: https://doi.org/10.1007/978-88-470-2278-2_75
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