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Zusammenfassung

Die Annahme, daβ oxidativer Streβ im Rahmen der Ätiopathogenese der Alzheimer Krankheit (DAT) eine wichtige Rolle spielen könnte, wird heute als vergleichsweise gut abgesicherte Hypothese angesehen. Man knüpft dabei an überlegungen an, die im Zusammenhang mit natürlichen Alterungsvorgängen und anderen neurodegenerativen Krankheiten schon intensiv erörtert worden sind. Besonders bei der Parkinsonschen Krankheit oder der amyotrophen Lateralsklerose wird dem Modell des neuronalen Zellunterganges durch oxidativen Streβ erhebliche Bedeutung zugesprochen (Hal-Liwell 1992, Jenner 1994, Frölich und Riederer 1995).

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© 1999 Springer-Verlag Wien

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Rösler, M., Retz, W. (1999). Therapie von oxidativem Streß. In: Riederer, P., Laux, G., Pöldinger, W. (eds) Neuro-Psychopharmaka Ein Therapie-Handbuch. Springer, Vienna. https://doi.org/10.1007/978-3-7091-6400-6_71

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  • DOI: https://doi.org/10.1007/978-3-7091-6400-6_71

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