Correction: Journal of Health, Population and Nutrition (2023) 42:74 https://doi.org/10.1186/s41043-023-00423-0


Following publication of the original article [1], Figs. 3 and 4 and Table 1 were found to contain overlap with a separate article [2]. The corrected figures and table are shown below.

Fig. 3
figure 3

Role of magnesium in various physiological and pathological consequences. Mg is associated with immune response (A), cancer progression (B), infectious diseases (C), and inflammation (D). Inflammation is induced by magnesium depletion via numerous signaling mechanisms [40, 103]. NMDA N-methyl-D-aspartate, RAAS the renin–angiotensin–aldosterone system

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Fig. 4
figure 4

Intracellular Mg deprivation induces oxidative stress including malfunction in mitochondria [40]. ATP adenosine triphosphate, ETC electron transport chain, F0F1-ATPases membrane-bound ATP synthases, IMAC inner membrane anion channel, KATP ATP-sensitive K channel, MnSOD manganese superoxide dismutase, MRS2 mitochondrial RNA splicing 2, PTP permeability transition pore, ROS reactive oxygen species, VDAC voltage-dependent anion channel

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Table 1 Relevance of Mg deficiency in various pathological abnormalities as investigated in animal model and clinical trials
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The updated version aligns with ethical guidelines and policies and includes sufficient reference to previous articles. The authors sincerely apologize for the errors. The errors do not affect the conclusion of the article.


Figures 3 and 4 and Table 1 have been updated in this correction and the original article [1] has been corrected.