Erratum

Unfortunately, after publication of the original version of this article [1], it was noticed that there were some errors in Fig. 3 and Fig. 4:

  • In Fig. 3, the methylation of H19/IGF2:IG-DMR hypomethylation is not correctly illustrated: the lolly pops should be empty (=unmethylated).

  • In Fig. 4, the methylation of both H19/IGF2:IG-DMR hypermethylation and KCNQ1OT1:TSS-DMR hypomethylation are not correctly illustrated: in case of the H19/IGF2:IG-DMR hypermethylation the lolly pops should be filled (=methylated), and for the KCNQ1OT1:TSS-DMR, they should be empty (=unmethylated).

The corrected Fig. 3 and Fig. 4 have been included in this erratum.

Fig. 3
figure 1

The 11p15.5 cluster can be divided in two functional domains whose imprinting is dependent on distinct imprinting control regions (H19/IGF2: IG DMR and KCNQ1OT1: TSS DMR). Mainly hypomethylation of the KCNQ1OT1: TSS DMR is responsible for SRS

Fig. 4
figure 2

Epimutations and mutations in 11p15.5 are also responsible for BWS