Correction: Genome Biol 24, 194 (2023)

https://doi.org/10.1186/s13059-023-03032-6

Following publication of the original article [1], the authors reported an error in Fig. 9, namely a missing significant difference symbol for JCR1 and a redundant significant difference symbol for JOE1. The updated Fig. 9 is available in this Correction.

Fig. 9
figure 1

Phenotype of GhLAX1, GhLAX2, GhLOX3 knock out and overexpression callus with hypocotyls as explants. a Schematic view of gRNA1, gRNA2 target sites in the GhLAX1, and GhLOX3 and overexpression cassette of GhLAX2. b Paraffin sections of hypocotyls infected with Agrobacterium after induction on callus induction medium for 0, 24, and 72 h. The red box represents the proliferation site. c The phenotypes of different transgenic explants and control (P7N) at 20 days post-induction and the callus proliferation rate (CPR) of explants and control at 20 days post-induction. d The phenotype of callus on the GhLAX1 knock out and GhLAX2 overexpression explants at about 70 days post-induction. Scale bar, 100 μm. e Days of embryonic callus occurrence of diferent transgenic explants. f Morphology of somatic cell embryos of JOE1. Scale bar, 100 μm

Additionally, the following text describing the experimental results shown in Fig. 9 has been amended as follows:

Previous text: Although all explants (CRISPR and overexpression) produced callus after 20 days of induction (Fig. 9c and Additional file 1: Figure. S11a), the callus proliferation rate (CPR) after 20 days of induction showed that the CPR of JCR1 was 58% and for JOE1, 130%, both significantly different to control (88%) in Jin668 (t-test, P < 0.05), suggesting that these LAX genes may play import roles in callus proliferation in Jin668. Notably, there was no significant difference between TCR1 and TP7N (71% Vs 77%; Fig. 9c).

Updated text: Although all explants (CRISPR and overexpression) produced callus after 20 days of induction (Fig. 9c and Additional file 1: Fig. S11a), the callus proliferation rate (CPR) after 20 days of induction showed that the CPR of JCR1 was 58% and for JOE1, 130%. JCR1 showed significantly different to control (88%) in Jin668 (t-test, P < 0.05), suggesting that these LAX genes may play import roles in callus proliferation in Jin668. Notably, there was no significant difference between TCR1 and TP7N (71% Vs 77%; Fig. 9c).

The original article has been updated.