Correction: J Exp Clin Cancer Res 38, 39 (2019)
https://doi.org/10.1186/s13046-019-1052-z
Following publication of the original article [1], an overlapping of images was identified Fig. 1e and Fig. 5b.
The images of Oct4 in TS and TS + DMSO groups were replaced and the correct Fig. 5 is given as below:
p38 suppression reversed long term TS exposure-triggered CSC-like properties. Mice exposed to TS were treated with or without p38 MAPK inhibitor (SB 203580) for 12 weeks, and representative micrographs of liver tissue were stained with H&E (a). b Immunohistochemical staining for EpCAM, CD133 and Nanog, Oct4 in liver tissues. c-f Fold changes of EpCAM (c), CD133 (d), Nanog (e) and Oct4 (f)—positive area in TS group compared with FA group. g Western blotting of EpCAM and CD133 in liver tissues. β-actin was served as the loading control. h-i The indicated proteins relative to β-actin were assessed by densitometric analysis; six animal samples per group were used for the densitometric analysis. Data are expressed as mean ± SD. The significance was assessed with one-way ANOVA test. # P < 0.05, ## P < 0.01, compared with FA control; * P < 0.05, ** P < 0.01, compared with TS + DMSO group. FA = filtered air; TS = tobacco smoke
The correction does not affect the overall results or conclusion of the article.
Reference
Xie C, Zhu J, Wang X, et al. Tobacco smoke induced hepatic cancer stem cell-like properties through IL-33/p38 pathway. J Exp Clin Cancer Res. 2019;38:39. https://doi.org/10.1186/s13046-019-1052-z.
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Xie, C., Zhu, J., Wang, X. et al. Correction: Tobacco smoke induced hepatic cancer stem cell-like properties through IL-33/p38 pathway. J Exp Clin Cancer Res 42, 141 (2023). https://doi.org/10.1186/s13046-023-02722-0
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DOI: https://doi.org/10.1186/s13046-023-02722-0