Correction: J Exp Clin Cancer Res 39, 128 (2020)
https://doi.org/10.1186/s13046-020-01638-3
Following publication of the original article [1], author identified an error in Fig. 2, specifically:
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Figure 2h - the cell migration assay of Hep3B-Vector group
The correct figure is presented below:
NCSTN promotes HCC cell growth and metastasis in vitro. a The effects of NCSTN knockdown and overexpression were examined by western blotting analysis in HCCLM3 and Hep3B cells. Loading control was assessed by β-actin. b CCK8 assays showed NCSTN depletion inhibited cell growth of HCCLM3 and NCSTN overexpression promoted cell growth of Hep3B. c, d Colony formation assays showed colony numbers in HCC cells with NCSTN depletion or overexpression. e, f The cell cycle assays showed that NCSTN depletion increased the G0/G1 fraction and decreased the S and G2/M fraction in HCCLM3 cells, whereas NCSTN overexpression decreased the G0/G1 fraction and increased the S and G2/M fraction in Hep3B cells. g, h The migration and invasion capacity was determined in the indicated HCC cells. Scale bar, 100 μm. i, j Wound healing assays showed the migration capacity of indicated HCC cells. Scale bar, 100 μm. HCC, hepatocellular carcinoma; CCK8, cell counting kit-8. *p < 0.05, **p < 0.01, ***p < 0.001
This correction does not change the result, interpretation, and conclusions of the study. The original article has been corrected.
Reference
Li H, Lan T, Xu L, et al. NCSTN promotes hepatocellular carcinoma cell growth and metastasis via β-catenin activation in a Notch1/AKT dependent manner. J Exp Clin Cancer Res. 2020;39:128. https://doi.org/10.1186/s13046-020-01638-3.
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Li, H., Lan, T., Xu, L. et al. Correction: NCSTN promotes hepatocellular carcinoma cell growth and metastasis via β-catenin activation in a Notch1/AKT dependent manner. J Exp Clin Cancer Res 42, 47 (2023). https://doi.org/10.1186/s13046-023-02617-0
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DOI: https://doi.org/10.1186/s13046-023-02617-0