Correction to: J Exp Clin Cancer Res 36, 172 (2017)
https://doi.org/10.1186/s13046-017-0635-9
Following publication of the original article [1], minor errors we identified in Fig. 6d and Fig. S4; specifically:
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Figure 6d: incorrect image used for shCtrl scratch image (24 h); correct image now used
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Fig. S4: incorrect images used for the invasive MDA-MB-231 cells (25 μM EA); correct images now used
ACTN4 promotes breast cancer proliferation and metastasis in vitro and in vivo. a Intracellular expression of ACTN4 was determined by Western blot (left) and real-time PCR (right) analysis, respectively; b ACTN4 expression was modified by transfecting recombinant plasmid or its shRNA in breast cancer cells and subjected to Western blotting (left) and real-time PCR (right) validation; c MTT and colony formation assay showed that ACTN4 silencing abrogated breast cancer cell proliferation while its overexpression promoted cell growth; d ACTN4 silencing inhibited the migration and invasion abilities of MDA-MB-231 cells; e ACTN4 silencing inhibited breast cancer growth and lung metastasis in vivo (*P < 0.05, **P < 0.01 versus control, values represented as the mean ± SD, n = 3)
The corrected figures, produced using the original data, are given here. The correction does not have any effect on the final conclusions of the paper.
Change history
07 April 2022
An amendment to this paper has been published and can be accessed via the original article.
Reference
Wang N, Wang Q, Tang H, et al. Direct inhibition of ACTN4 by ellagic acid limits breast cancer metastasis via regulation of β-catenin stabilization in cancer stem cells. J Exp Clin Cancer Res. 2017;36:172. https://doi.org/10.1186/s13046-017-0635-9.
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Additional file 4.
The wound healing and chamber invasive assay revealed that breast cancer cell migration and invasion were inhibited by EA in a time- and dose-dependent manner.
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Wang, N., Wang, Q., Tang, H. et al. Correction to: Direct inhibition of ACTN4 by ellagic acid limits breast cancer metastasis via regulation of β-catenin stabilization in cancer stem cells. J Exp Clin Cancer Res 41, 118 (2022). https://doi.org/10.1186/s13046-022-02341-1
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DOI: https://doi.org/10.1186/s13046-022-02341-1