Correction to: J Exp Clin Cancer Res 38, 475 (2019)
https://doi.org/10.1186/s13046-019-1433-3
Following publication of the original article [1], the authors identified some minor errors in image-typesetting in Fig. 2; specifically in Fig. 2e.
The corrected figure is given below. The correction does not have any effect on the results or conclusions of the paper.
Overexpression of FOXG1 promotes cell mobility and invasion by inducing epithelial-mesenchymal transition (EMT). a Western blotting analysis and b Immunofluorescence analysis of expression of epithelial cell markers (E-cadherin and α-catenin) and mesenchymal cell markers (vimentin and fibronectin) in indicated cells transfected with FOXG1 expression vector or control vector. Nuclei counterstained with DAPI. GAPDH was used as a loading control. c Representative migrating images of the indicated Huh7 and PLC/PRF5y cells on uncoated Transwell devices in five random fields. d Quantification of the invading cells of the indicated Huh7 and PLC/PRF5y cells on Matrigel-coated Transwell devices in five random fields. Values represent mean ± SD. *P < 0.05. e Representative micrographs of wound healing assay of the indicated Huh7 and PLC/ PRF5y cells. Wound closures were photographed at 0, 24, and 36 h after wounding. All experiments were repeated at least three times with similar results
Reference
Zheng X, Lin J, Wu H, et al. Forkhead box (FOX) G1 promotes hepatocellular carcinoma epithelial-Mesenchymal transition by activating Wnt signal through forming T-cell factor-4/Beta-catenin/FOXG1 complex. J Exp Clin Cancer Res. 2019;38:475 https://doi.org/10.1186/s13046-019-1433-3.
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The article to which this update relates has been retracted. Please see the retraction notice for more detail: https://doi.org/10.1186/s13046-023-02739-5
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Zheng, X., Lin, J., Wu, H. et al. Correction to: Forkhead box (FOX) G1 promotes hepatocellular carcinoma epithelial-Mesenchymal transition by activating Wnt signal through forming T-cell factor-4/Betacatenin/FOXG1 complex. J Exp Clin Cancer Res 40, 104 (2021). https://doi.org/10.1186/s13046-021-01900-2
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DOI: https://doi.org/10.1186/s13046-021-01900-2