Correction to: Journal of Neuroinflammation (2019) 16:2 https://doi.org/10.1186/s12974-018-1383-2
The original version [1] of the article unfortunately contained mistakes in Figs. 5 and 6. The mistake occurred due to a copying and labeling error with image selection; all analyses remain the same.
It has been corrected in this correction (Figs. 5 and 6).
Astrocytes labelled with GFAP antibody in the frontal cortex and hippocampus. A A significant reduction in GFAP-positive cells was seen in TBI + MSCs (***p < 0.001) and TBI + MSCs + IL-10 (***p < 0.001) groups in the frontal cortex in comparison to TBI + vehicle group. B In the CA1 region of the hippocampus, a significant reduction of GFAP positive cells were found in the TBI + MSCs + IL-10 (***p < 0.001) and sham + vehicle group (*p < 0.05) in comparison to TBI + vehicle group. C In the CA3 region of the hippocampus, TBI + vehicle group had a significant increase in GFAP-positive cells when compared to all other groups (*p < 0.05, **p < 0.01; scale bar = 50 µm). Error bars represent ± SEM
Macrophages/microglia labelled with Iba1 antibody in the frontal cortex and hippocampus. A In the frontal cortex an injury effect was seen with an increased number of Iba1 positive cells in all TBI groups (*p < 0.05, ***p < 0.001). B No significant differences were seen among the groups in the CA1 region of the hippocampus. C No significant differences were seen among the groups in the CA3 region of the hippocampus (scale bar = 50 µm). Error bars represent ± SEM
Reference
Peruzzaro ST, Andrews MM, Al-Gharaibeh A, Pupiec O, Resk M, Story D, Maiti P, Rossignol J, Dunbar GL. Transplantation of mesenchymal stem cells genetically engineered to overexpress interleukin-10 promotes alternative inflammatory response in rat model of traumatic brain injury. J Neuroinflamm. 2019;16:2.
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Peruzzaro, S.T., Andrews, M.M.M., Al-Gharaibeh, A. et al. Correction to: Transplantation of mesenchymal stem cells genetically engineered to overexpress interleukin-10 promotes alternative inflammatory response in rat model of traumatic brain injury. J Neuroinflammation 19, 15 (2022). https://doi.org/10.1186/s12974-021-02362-1
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DOI: https://doi.org/10.1186/s12974-021-02362-1