Anterior shoulder dislocations, unlike posterior, happen, almost always, secondary to trauma. Posterior shoulder dislocations usually occur following unbalanced muscle contractions (electric shock, epileptic seizure etc) [5, 8]. The reason why the shoulder dislocates anteriorly after trauma is that as the arm extends and abducts, impingement of the greater tuberosity on the acromion levers the humeral head out of the glenoid . Moreover the rotator cuff pushes downwards the humeral head which is finally displaced anteriorly by the flexors and external rotators. The posterior dislocations are more common after seizure since the contraction of the relatively weak external rotators and the posterior fibers of the deltoid are overcome by the more powerful internal rotator. The succeeding adduction and internal rotation usually causes the humeral head to dislocate posteriorly . One suggestion about bilateral anterior dislocation following a seizure is that this may occur not during the muscle contractions but from the trauma of the shoulders striking the floor, after the collapse . Bilateral occurrence of anterior shoulder dislocation is rare because almost always one extremity takes the brunt of the impact during the trauma incidence [5, 8]. In our report we may suggest that loss of consciousness after the seizure did not allow the patient to react and reflexly protect one of his arms by exposing the other.
Associated fracture of the greater tuberosity occurs in 15% of the anterior dislocation cases and indicates an associated rotator cuff tear. If the greater tuberosity fracture is displaced the diagnosis of a rotator cuff tear is almost certain . This may cause long term instability and functional impairment if the fragment is not anatomically reduced . Thus internal fixation after the reduction must be the rule in such cases.
Closed reduction of a neglected anterior shoulder dislocation can be performed only up to six weeks post injury. After this period the danger of an iatrogenic fracture or neurovascular damage raises too high and operative procedures shall be followed. 
In our case the 3 weeks interval between the injury and the rehabilitation did not seem to influence the final functional outcome. Shoulder stabilization was not performed but the patient experienced no recurrent dislocation after two years of follow-up. As the shoulder is a muscle dependant joint one could suggest that when rotator cuff tears can be excluded, a proper physiotherapy program of muscle enforcement alone could be sufficient for a very good functional result, even in neglected cases like this one.
The literature suggests that over 10% of documented bilateral anterior shoulder dislocations following trauma were diagnosed late . As there is a greater awareness of anterior shoulder dislocations for trauma patients, it would be reasonable to assume that there may be a higher incidence of delayed diagnosis of such an injury following a presentation with an indirect complaint, such as a seizure. The unusual presentation combined with the patient's post-ictal discomfort and drowsy state may potentially delay the diagnosis. As this could affect the prognosis, early recognition is vital.