Correction

In the published work [1], Figure three A panels e and f and Figure four B (Figure 1B here) panels b and f represent the same types of samples in two different experiments, i.e., CIP4 siRNA-transfected myocytes cultured in the absence and presence of phenylephrine, respectively. However, in the original version of Rusconi, et al. [1], the panels in Figure three A e and f were unintentionally duplicated in the panels in Figure four B (Figure 1B here) b and f, respectively. In this correction, a new Figure four (Figure 1 here) is provided with different panels for figure four B (Figure 1B) b and f. The interpretation and conclusion of the depicted experiments remain the same.

Figure 1
figure 1

The CIP4 FCH domain is important for neonatal rat ventricular myocyte hypertrophy. Neonatal rat ventricular myocytes were transfected with control or CIP4 siRNA and then infected with adenovirus expressing myc-tagged CIP4 WT or ΔFCH protein. Myocytes were stimulated with 10 μM PE for two days as indicated. A. CIP4 proteins were detected using a mouse anti-CIP4 antibody against human CIP4 aa 411–501. (Rat and human CIP4 are 92% identical.) B. Immunocytochemistry for α-actinin (green), ANF (red) and Hoechst (blue); bar = 20 μm. C. Cross-section area of myocytes. n = 7. D. Fraction of myocytes expressing ANF. n = 6. ANOVA (two-factor with replication): p-value (among the four CIP4 expression conditions) = 0.005 (C) and = 0.02 (D); p-value (±PE) < 10-6 for both B and C. Post-hoc testing: *p-values vs. CIP4 siRNA-transfected myocytes; †p-values comparing myocytes cultured ± PE; ‡p-values vs. myc-CIP4 WT expressing myocytes.

Full size image

Corrected Figure four (Figure 1 here):