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Opsoclonus associated with autoantibodies to glutamate receptors δ2

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Fig. 1

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Acknowledgments

This study was funded by a grant from Scientific Research (C) (No. 23590869 and No. 26460901) from the Ministry of Education, Culture, Sports, Scinence and Technology of Japan.

Conflict of interest

The authors declare no conflicts of interest.

Ethical approval

All procedures performed in studies involving human participants were in accordance with the ethical standards of the institutional and/or national research committee and with the 1964 Helsinki declaration and its later amendments or comparable ethical standards.

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Authors and Affiliations

  1. Division of Clinical Medicine, Department of Neurology, Faculty of Medicine, University of Tsukuba, Tsukuba, Ibaraki, 305-8575, Japan

    Takashi Hosaka, Kiyotaka Nakamagoe, Naomi Mamada & Akira Tamaoka

  2. National Epilepsy Center, Shizuoka Institute of Epilepsy and Neurological Disorders, 886 Urushiyama, Aoi-ward, Shizuoka, Japan

    Yukitoshi Takahashi

Authors
  1. Takashi Hosaka
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  2. Kiyotaka Nakamagoe
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  3. Yukitoshi Takahashi
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  4. Naomi Mamada
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  5. Akira Tamaoka
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Corresponding author

Correspondence to Kiyotaka Nakamagoe.

Electronic supplementary material

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Supplementary material 1 The patient presented with rapid, irregular eye movements in both the horizontal and vertical plane. (MPG 15332 kb)

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Hosaka, T., Nakamagoe, K., Takahashi, Y. et al. Opsoclonus associated with autoantibodies to glutamate receptors δ2. Neurol Sci 36, 1741–1742 (2015). https://doi.org/10.1007/s10072-015-2264-4

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  • DOI: https://doi.org/10.1007/s10072-015-2264-4

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