The resilient synapse: insights fromgenetic interference with synaptic cell adhesion molecules

Table 1 was rendered incorrectly: the phenotype details and reference for neuroligin mistakenly appeared on the line for β-neurexins. The corrected table is reproduced in full here.

Table 1 Phenotype of genetic lack-of-function models of important SCAMs (SCAMs synaptic cell adhesion molecules. CNS central nervous system, PNS peripheral nervous system, NMJ neuromuscular junction, PSD95 postsynaptic density protein 95, LTP long-term potentiation, HSPG heparan sulphate proteoglycans, AChR acetylcholine receptor, NMDAR N-methyl-D-aspartate receptor, TGF-β transforming growth factor β, GABA γ-aminobutyric acid). All phenotypes refer to cited mouse models except when explicitly stated otherwise