Impact of Waterborne Copper on the Structure of Gills and Hepatopancreas and its Impact on the Content of Metallothionein in Juvenile Giant Freshwater Prawn Macrobrachium rosenbergii (Crustacea: Decapoda)

Erratum to: Arch Environ Contam Toxicol 52(1):73–79 DOI 10.1007/s00244-005-0214-5

An incorrect version of Fig. 1 was printed in this paper. The corrected version is reprinted on the following page.

Fig. 1

Effects of waterborne copper on the microstructure of gills from juvenile Macrobrachium rosenbergii. a Typical organization of the gills (control). Gill lamellae (L) with uniform interlamellar space (ILS), ×200; b Typical organization of the gills (control). Normal hemocoelic space with the optimum number of hemocytes (HC) and pillar cells (PC) were found to extend into the lamellar sinus, ×400; c Exposure to 0.01 mg/L copper. Swelling of gill lamellae (SL), lifting of lamellar epithelium (LLE) and the accumulation of hemocytes (HC) in the hemocoelic space of the gill lamellae, ×400; d Exposure to 0.05 mg/L copper. Hemocytic infiltration (HI) in the hemocoelic space and fusion of gill lamellae (FL) in test prawns, ×400; e Exposure to 0.1 mg/L copper. Narrower (N) or obstructed hemolymph vessels and fusion of gill lamellae (FL) in test prawns, ×400; f Exposure to 0.2 mg/L copper. Necrotic gill lamellae (NL) and lifting of lamellar epithelium (LLE) in test prawns, ×400; g Exposure to 0.3 mg/L copper. Abnormal gill tips (AG) and malformation (MF) at the tips of the gills in test prawns, ×400. h Exposure to 0.4 mg/L copper. Fusion of gill lamellae (FL), swelling of gill lamellae (SL), and necrotic gill lamellae (NL) in test prawns, ×200