Abstract
Morphological examination of primary foci several hours after intramuscular infection of rats withPseudomonas aeruginosa, Staphylococcus aureus, orStaphylococcus epidermidis showed that bacteriopyknosis is associated with deficiency of compounds necessary for the microorganisms or excess of secreted microbial metabolites. Motility is a factor of virulence, since it inhibits bacteriopyknosis. The biological significance of divalent antibodies and agglutination provided by them probably lies in considerable stimulation of bacteriopyknosis in agglutinates. Formation of IgM also enhances bacteriopyknosis.
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Translated fromByulleten' Eksperimental'noi Biologii i Meditsiny, Vol. 123 No. 3, pp. 349–352, March, 1997
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Pal'tsyn, A.A., Kolokol'chikova, E.G., Badikova, A.K. et al. On the mechanisms of bacteriopyknosis. Bull Exp Biol Med 123, 303–306 (1997). https://doi.org/10.1007/BF02445435
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DOI: https://doi.org/10.1007/BF02445435