Abstract
A defect in the regulation of the phagocytic component of immunity is revealed in patients with atopic dermatitis with more than 70% skin involvement. This defect manifests itself in the lack of production of stimulating neutrophilokines and impaired receptor capacity of monocytes. The regulation of not only peripheral cells, but also their bone marrow precursors was impaired.
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Translated fromByulleten' Eksperimental'noi Biologii i Meditsiny, Vol. 120, No 9, pp 318–319, September, 1995
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Yarovinskii, B.G., Zurochka, A.V., Ebert, L.Y. et al. The regulatory function of neutrophils in patients with atopic dermatitis. Bull Exp Biol Med 120, 957–958 (1995). https://doi.org/10.1007/BF02445029
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DOI: https://doi.org/10.1007/BF02445029