Abstract
Acute pulmonary edema (PE) occurs when the pulmonary lymphatics fail to remove transupdated fluid [1]. The edema develops as fluid moves from the intravascular compartment into the interstitial space and from there, in severe cases, into the alveoli and eventually forms overt and copious pink frothy sputum. The most common causes of cardiogenic PE are in association with acute myocardial infarction (AMI) and the so called “flash” cardiogenic PE. While it is easy to understand how PE occurs in association with acute left ventricular failure (LVF) after AMI, the pathophysiology of “flash” PE is not clear. Moreover, the treatment of cardiogenic PE should be according to the cause of the edema. Current recommendations for treatment may not always be appropriate.
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References
Nakahara K, Nanjo S, Maeda M, Kawashima Y (1983) Dynamic insufficiency of lung lymph flow from the right lymph duct in dogs with acute filtration edema. Am Rev Respir Dis 127:67–71
Gaar KA Jr, Taylor AE, Owens LJ, Gayton AC (1967) Effect of capillary pressure and plasma protein on development of pulmonary edema. Am J Physiol 213:79–82
Sniderman A, Burdon T, Homan J, Salerno T (1984) Pulmonary blood flow. J Thorac Car-diovasc Surg 87:130–135
Timmis AD, Fowler MB, Burwood RJ, Gishen P, Vincent R, Chamberlain DA (1981) Pulmonary edema without critical increase in left atrial pressure in acute myocardial infarction. Br Med J 283:636–638
Soufer R, Wohlgelernter D, Vita NA, et al. (1985) Intact systolic left ventricular function in clinical congestive heart failure. Am J Cardiol 55:1032–1036
Altschule MD (1986) Acute pulmonary edema without demonstrable left ventricular failure after myocardial infarction. Chest 89:292–293
Dougherty AH, Naccarelli GV, Gray EL, Hicks CH, Goldstein RA (1984) Congestive heart failure with normal systolic function. Am J Cardiol 54:778–782
Wise RA (1986) Effect of circulatory mechanics on hydrostatic forces producing pulmonary edema. J Crit Care 1:247–255
Cabanes LR, Weber SN, Matran R, et al. (1984) Bronchial hyperresponsiveness to metha-choline in patients with impaired left ventricular function. New Engl J Med 320:1317–1322
Forrester AL, Cabin HS, Francis CK (1988) The syndrome of flash pulmonary edema: Clinical definition and angiographie findings. J Am Coll Cardiol 11:151A
Takayama Y, Iwasaka T, Sugiura T, et al. (1991) Increased extravascular lung water in patients with low pulmonary artery occlusion pressure after acute myocardial infarction. Crit Care Med 19:21–25
Haustein KO (1990) Review: Therapeutic concepts of congestive heart failure. Int J Clin Pharmacol Ther Toxicol 28:273–281
Perret C (1990) Acute heart failure in myocardial infarction: Principles of treatment. Crit Care Med 18:26–29
Braunwald E (1988) Pathophysiology of heart failure. In: Braunwald E (ed) Heart Disease-a textbook of cardiovascular medicine, 3rd edn. WB Saunders Philadelphia, pp 426–448
Navarrete-Navarro P, Vazquez G, Fernandez E, Torres JM, Reina A, Hinojosa R (1989) Mediastinal, left and right atrial pressure variations with different modes of mechanical and spontaneous ventilation. Crit Care Med 17:563–567
Rasanen J, Vaisanen IT, Heikkila J, Nikki P (1985) Acute myocardial infarction complicated by left ventricular dysfunction and respiratory failure. Chest 87:158–162
Vaisanen IT, Rasanen J (1987) Continuous positive airway pressure and supplemental oxygen in the treatment of cardiogenic pulmonary edema. Chest 92:481–485
Rasanen J, Heikkila J, Downs J, Nikki P. Vaisanen I, Viitanen A (1985) Continuous positive airway pressure by face mask in acute cardiogenic pulmonary edema. Am J Cardiol 55:296–300
Poulton EP, Oxon DM (1936) Left-sided heart failure with pulmonary oedema. Lancet 2:981–983
Hillman K, Huggins C (1991) A new continuous positive airway pressure (CPAP) device. Anaesth Intensive Care 19:233–260
Mathru M, Venus B, Smith RA (1985) All positive airway pressures are not created equal. Chest 87:137–138
Rasanen J (1987) Conventional and high frequency controlled mechanical ventilation in patients with left ventricular dysfunction and pulmonary edema. Chest 91:225–229
Crimi G, Conti G, Bufi M, et al. (1988) High frequency jet ventilation (HFJV) has no better hemodynamic tolerance than controlled mechanical ventilation (CMV) in cardiogenic shock. Intensive Care Med 14:359–363
Aubier M, Trippenbach T, Roussos C (1981) Respiratory muscle fatigue during cardiogenic shock. J Appl Physiol 51:499–508’
Miller LW, Merkle EJ, Herrmann V (1990) Outpatient dobutamine for end-stage congestive heart failure. Crit Care Med 18:30–34
Fedullo AJ, Swinburne AJ, Wahl GW, Bixby KR (1988) APACHE II score and mortality in respiratory failure due to cardiogenic pulmonary edema. Crit Care Med 16:1218–1221
Kerridge RK, Glasziou PP, Hillman KM (1992) The use of ‘quality-adjusted life years’ (QALYs) to evaluate treatment in intensive care (in press)
Daffurn K, Hillman KM (1992) Home management of cardiogenic pulmonary oedema with CPAP (in press)
Guyton AC (ed) Textbook of Medical Physiology, 8th edn. WB Saunders Co, Philadelphia
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Hillman, K. (1992). Acute Cardiogenic Pulmonary Edema. In: Vincent, JL. (eds) Yearbook of Intensive Care and Emergency Medicine 1992. Yearbook of Intensive Care and Emergency Medicine, vol 1992. Springer, Berlin, Heidelberg. https://doi.org/10.1007/978-3-642-84734-9_18
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DOI: https://doi.org/10.1007/978-3-642-84734-9_18
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