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Potassium Homeostasis in Obstructive Uropathy and Interstitial Nephritis

  • Chapter
Tubulo-Interstitial Nephropathies

Part of the book series: Developments in Nephrology ((DINE,volume 31))

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Abstract

Potassium homeostasis can be perturbed with both obstructive uropathy and interstitial nephritis. Hypokalemia can develop during the diuretic phase that accompanies relief of urinary tract obstruction and also been reported in association with acute interstitial nephritis. More typically, hyperkalemia is found in patients with chronic obstructive uropathy and is observed frequently in patients with Tubolointerstitial nephropathies. Both hyporeninemic hypoaldosteronism and primary defects in tubular secretion of potassium account for the development of hyperkalemia. Hyperchloremic acidosis (Type 4 RTA) frequently accompanies these hyperkalemic syndromes. Current concepts regarding the pathophysiology of both hyperkalemia and Type 4 RTA are considered.

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Author information

Authors and Affiliations

  1. Department of Medicine, University of Southern California, Los Angeles, CA, USA

    Richard L. Tannen

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  1. Richard L. Tannen
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Editor information

Editors and Affiliations

  1. University of Bari, Bari, Italy

    Alberto Amerio  & Pasquale Coratelli  & 

  2. University of Southern California, Los Angeles, California, USA

    Shaul G. Massry

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© 1991 Springer Science+Business Media New York

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Tannen, R.L. (1991). Potassium Homeostasis in Obstructive Uropathy and Interstitial Nephritis. In: Amerio, A., Coratelli, P., Massry, S.G. (eds) Tubulo-Interstitial Nephropathies. Developments in Nephrology, vol 31. Springer, Boston, MA. https://doi.org/10.1007/978-1-4615-3892-9_19

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  • DOI: https://doi.org/10.1007/978-1-4615-3892-9_19

  • Publisher Name: Springer, Boston, MA

  • Print ISBN: 978-1-4613-6736-9

  • Online ISBN: 978-1-4615-3892-9

  • eBook Packages: Springer Book Archive

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