Abstract
Pain is defined by the International Association for the Study of Pain as “an unpleasant sensory and emotional experience associated with actual or potential tissue damage.” Caregivers involved in pain management suggest that pain and the intensity of discomfort are whatever the patient states and should be managed accordingly.
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References
Ji RR, Woolf CJ. Neuronal plasticity and signal transduction in nociceptive neurons: implications or the initiation and maintenance of pathological pain. Neurobiol Dis. 2001;8(1):1–10.
Treed RD, Meyer RA, Raja SN, Campbell JN. Peripheral and ventral mechanisms of cutaneous hyperalgesia. Prog Neurobiol. 1992;38(4):397–421.
Wang H, Kohno T, Amaya F, et al. Bradykinin produces pain hypersensitivity by potentiation spinal cord glutamatergic synaptic transmission. J Neurosci. 2005;25(35):7986–92.
Woolf CJ. Evidence for a central component of post-injury pain hypersensitivity. Nature. 1983;306(5944):686–88.
Woolf CJ, Mannion RJ. Neuropathic pain: etiology, symptoms, mechanisms, and management. Lancet. 1999;353(9168):1959–64.
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Mirchandani, A., Saleeb, M., Sinatra, R. (2011). Acute and Chronic Mechanisms of Pain. In: Vadivelu, N., Urman, R., Hines, R. (eds) Essentials of Pain Management. Springer, New York, NY. https://doi.org/10.1007/978-0-387-87579-8_4
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DOI: https://doi.org/10.1007/978-0-387-87579-8_4
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