Abstract
Recent evidence that insoluble aggregates of beta-amyloid peptide in the brains of Alzheimer patients may elicit neurodestructive immune responses offers the prospect of new therapies to treat this neurodegenerative condition.
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Pachter, J. Inflammatory mechanisms in Alzheimer disease: the role of beta-amyloid/glial interactions. Mol Psychiatry 2, 91–95 (1997). https://doi.org/10.1038/sj.mp.4000212
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DOI: https://doi.org/10.1038/sj.mp.4000212
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