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Similar patterns of chromosome abnormalities in CML occur in addition to the Philadelphia chromosome with or without tyrosine kinase inhibitor treatment

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References

  1. Cortes J, O′Dwyer ME . Clonal evolution in chronic myelogenous leukemia. Hematol Oncol Clin North Am 2004; 18: 671–684.

    Article  Google Scholar 

  2. Johansson B, Fioretos T, Mitelman F . Cytogenetic and molecular genetic evolution of chronic myeloid leukemia. Acta Haematol 2002; 107: 76–94.

    Article  CAS  Google Scholar 

  3. Cortes JE, Talpaz M, Giles F, O′Brien S, Rios MB, Shan J et al. Prognostic significance of cytogenetic clonal evolution in patients with chronic myelogenous leukemia on imatinib mesylate therapy. Blood 2003; 101: 3794–3800.

    Article  CAS  Google Scholar 

  4. O′Dwyer ME, Mauro MJ, Kurilik G, Mori M, Balleisen S, Olson S et al. The impact of clonal evolution on response to imatinib mesylate (STI571) in accelerated phase CML. Blood 2002; 100: 1628–1633.

    Article  Google Scholar 

  5. O'Dwyer ME, Mauro MJ, Blasdel C, Farnsworth M, Kurilik G, Hsieh YC et al. Clonal evolution and lack of cytogenetic response are adverse prognostic factors for hematologic relapse of chronic phase CML patients treated with imatinib mesylate. Blood 2004; 103: 451–455.

    Article  CAS  Google Scholar 

  6. Hiller B, Bradtke J, Balz H, Rieder H . CyDAS: a cytogenetic data analysis system. Bioinformatics 2005; 21: 1282–1283.

    Article  CAS  Google Scholar 

  7. Karrman K, Sallerfors B, Lenhoff S, Fioretos T, Johansson B . Cytogenetic evolution patterns in CML post-SCT. Bone Marrow Transplant 2007; 39: 165–171.

    Article  CAS  Google Scholar 

  8. Chase A, Pickard J, Szydlo R, Coulthard S, Goldman JM, Cross NC . Non-random involvement of chromosome 13 in patients with persistent or relapsed disease after bone-marrow transplantation for chronic myeloid leukemia. Genes Chromosomes Cancer 2000; 27: 278–284.

    Article  CAS  Google Scholar 

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Authors and Affiliations

  1. MLL Munich Leukaemia Laboratory, Munich, Germany

    C Haferlach, S Schnittger, T Weiss, W Kern & T Haferlach

  2. Clinic for Stem Cell Transplantation, University Cancer Center Hamburg, Hamburg, Germany

    U Bacher

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  1. C Haferlach
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  2. U Bacher
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  3. S Schnittger
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  4. T Weiss
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  5. W Kern
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  6. T Haferlach
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Correspondence to C Haferlach.

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Haferlach, C., Bacher, U., Schnittger, S. et al. Similar patterns of chromosome abnormalities in CML occur in addition to the Philadelphia chromosome with or without tyrosine kinase inhibitor treatment. Leukemia 24, 638–640 (2010). https://doi.org/10.1038/leu.2009.222

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