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Tenth annual meeting of the Italian Network for Tumor Biotherapy (NIBIT), SIENA, Italy, November 5–7, 2012

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Abbreviations

1-MT:

1-Methyl tryptophan

AML:

Acute myeloid leukemia

ACT:

Adoptive cell therapy

AEs:

Adverse events

Haplo-HSCT:

Haploidentical hematopoietic stem cell transplantation

APC:

Antigen-presenting cells

APM:

Antigen processing and presentation machinery

B-NHLs:

B cell non-Hodgkin lymphoma

BLI:

Bioluminescence imaging

BM:

Bone marrow

CIC:

Cancer initiating cells

CSC:

Cancer stem cells

CTA:

Cancer–testis antigen

CEA:

Carcinoembryonic antigen

CCL:

Chemokine (C–C motif) ligand

CAR:

Chimeric antigen receptors

CLL:

Chronic lymphocytic leukemia

CRC:

Colorectal cancer cells

CREB:

cAMP response element-binding protein

CIK:

Cytokine-induced killer

CTLA-4:

Cytotoxic T-lymphocyte antigen 4

DC:

Dendritic cells

T-bodies:

Engineered T cells

EAP:

Expanded access program

GVHD:

Graft versus host disease

VH CDR3:

Heavy complementarity-determining region 3

HLA:

Human leukocyte antigen

hPSMA:

Human prostate-specific membrane antigen

HAS:

Human serum albumin

Ig:

Immunoglobulin

IDO:

Indoleamine 2,3-dioxygenase

isoDGR:

IsoAsp-Gly-Arg

KIR:

Killer cell immunoglobulin-like receptor

LAG-3:

Lymphocyte activation gene-3

LN:

Lymph node

MHC:

Major histocompatibility complex

MC:

Mast cells

MMP-9:

Metalloproteinase-9

mAb:

Monoclonal antibody

MM:

Multiple myeloma

MDSC:

Myeloid-derived suppressor cells

NK:

Natural killer

NSCLC:

Non-small-cell lung cancer

OS:

Overall survival

PBMC:

Peripheral blood mononuclear cell

PD-1:

Programmed cell death-1

PD-L1:

Programmed cell death-1 ligand 1

TGF:

Transforming growth factor

TRAMP:

Transgenic adenocarcinoma of the mouse prostate

TAA:

Tumor-associated antigen

TNF-alpha:

Tumor necrosis factor alpha

TCR:

T cell receptor

Treg:

Regulatory T cells

CD44v6:

Variant 6 of the adhesion receptor CD44

VEGF:

Vascular endothelial growth factor

Acknowledgments

This work was supported in part by grants from: Associazione Italiana per la Ricerca sul Cancro (Hugues Jean Marie Nicolay fellow) and Istituto Toscano Tumori. A special thanks to the NIBIT members and meeting speakers for their contribution to the manuscript and the conference.

Conflict of interest

The authors declare that they have no conflict of interest.

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Correspondence to Michele Maio.

Additional information

This study was conducted on behalf of Italian Network for Tumor Biotherapy (NIBIT), Siena, Italy.

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Maio, M., Nicolay, H.J.M., Ascierto, P.A. et al. Tenth annual meeting of the Italian Network for Tumor Biotherapy (NIBIT), SIENA, Italy, November 5–7, 2012. Cancer Immunol Immunother 62, 1851–1858 (2013). https://doi.org/10.1007/s00262-013-1496-5

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  • DOI: https://doi.org/10.1007/s00262-013-1496-5

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