Abstract
A gap, split, or defect in the midline of roof of mouth communicating both oral and nasal cavities at birth is referred to as cleft palate. Many genetic factors and environmental teratogens (maternal usage of corticosteroids, tretenoins, alcohol, anticonvulsants, and smoking during pregnancy) are assumed to be linked with development of cleft palate (LaRossa et al. 2010; Dixon 2011; Kosowski et al. 2012; Leslie and Marazita 2013).
It is categorized as anterior and posterior cleft palate depending upon lack of fusion anterior or posterior to incisive foramen. Cleft of posterior palate may also present as bifid uvula, cleft of soft palate, and cleft of soft and hard palate. A complete cleft involves both anterior and posterior palate. Patients of cleft palate may present with difficulty in feeding coupled with reflux of food through the nose. Babies are often malnourished as they are unable to suck milk adequately. If not repaired timely, child will develop delayed and improper speech, recurrent ear infection, and dental problems.
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Sohail, M., Bashir, M.M. (2022). Anatomy of Cleft Palate. In: Fayyaz, G.Q. (eds) Surgical Atlas of Cleft Palate and Palatal Fistulae. Springer, Singapore. https://doi.org/10.1007/978-981-15-3889-6_3-1
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DOI: https://doi.org/10.1007/978-981-15-3889-6_3-1
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