Definition
The ability of nutritional factors (including food, alcohol, nutritional status, and physical activity) to increase or decrease the risk of colorectal cancer.
Characteristics
The etiology of colorectal cancer is multifactorial. Generally, it results from an interaction between genetic and environmental factors. As a part of the digestive tract, the colon is exposed to many native or metabolized dietary compounds. Gut microbiota, which metabolizes various dietary constituents and modifies their bioavailability and effects on the host, is also involved in colon carcinogenesis. The development of colon cancer is modulated by several nutritional factors, some acting as risk factors and others as protective factors.
Relation Between Nutrition and Colon Cancer: Evaluation by an International Expert...
References
Baan R, Straif K, Grosse Y et al (2007) Carcinogenicity of alcoholic beverages. Lancet Oncol 8:292–293
Calle EE, Kaaks R (2004) Overweight, obesity and cancer: epidemiological evidence and proposed mechanisms. Nat Rev Cancer 4:579–591
Fridenreich CM, Orenstein MR (2002) Physical activity and cancer prevention: etiologic evidence ad biological mechanisms. J Nutr 132:3456S–3464S
WCRF/AICR (2011) Continuous Update Project Report. Food, Nutrition, Physical Activity, and the Prevention of Colorectal Cancer. WCRF/AICR, Washington, DC. 40 pages
WCRF/AICR (2009) Policy and action for cancer prevention. Food, nutrition, and physical activity: a global perspective. AICR, Washington, DC. 190 pages
See Also
(2012) Acetaldehyde. In: Schwab M (ed) Encyclopedia of cancer, 3rd edn. Springer, Berlin/Heidelberg, p 16. doi:10.1007/978-3-642-16483-5_22
(2012) Alcohol dehydrogenase. In: Schwab M (ed) Encyclopedia of cancer, 3rd edn. Springer, Berlin/Heidelberg, p 120. doi:10.1007/978-3-642-16483-5_6732
(2012) Alcohol-mediated cancer. In: Schwab M (ed) Encyclopedia of cancer, 3rd edn. Springer, Berlin/Heidelberg, p 126. doi:10.1007/978-3-642-16483-5_170
(2012) Carcinogen. In: Schwab M (ed) Encyclopedia of cancer, 3rd edn. Springer, Berlin/Heidelberg, p 644. doi:10.1007/978-3-642-16483-5_839
(2012) Confounding. In: Schwab M (ed) Encyclopedia of cancer, 3rd edn. Springer, Berlin/Heidelberg, p 968. doi:10.1007/978-3-642-16483-5_1304
(2012) C-reactive protein. In: Schwab M (ed) Encyclopedia of cancer, 3rd edn. Springer, Berlin/Heidelberg, p 993. doi:10.1007/978-3-642-16483-5_1369
(2012) Folate. In: Schwab M (ed) Encyclopedia of cancer, 3rd edn. Springer, Berlin/Heidelberg, p 1440. doi:10.1007/978-3-642-16483-5_2231
(2012) Free radicals. In: Schwab M (ed) Encyclopedia of cancer, 3rd edn. Springer, Berlin/Heidelberg, p 1454. doi:10.1007/978-3-642-16483-5_2267
(2012) Insulin. In: Schwab M (ed) Encyclopedia of cancer, 3rd edn. Springer, Berlin/Heidelberg, pp 1873-1874. doi:10.1007/978-3-642-16483-5_3075
(2012) Leptin. In: Schwab M (ed) Encyclopedia of cancer, 3rd edn. Springer, Berlin/Heidelberg, p 2001. doi:10.1007/978-3-642-16483-5_6734
(2012) Proliferation. In: Schwab M (ed) Encyclopedia of cancer, 3rd edn. Springer, Berlin/Heidelberg, p 3004. doi:10.1007/978-3-642-16483-5_4766
(2012) Risk factor. In: Schwab M (ed) Encyclopedia of cancer, 3rd edn. Springer, Berlin/Heidelberg, p 3310. doi:10.1007/978-3-642-16483-5_5111
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Latino-Martel, P., Druesne-Pecollo, N., Hercberg, S., Touvier, M. (2014). Colorectal Cancer Nutritional Carcinogenesis. In: Schwab, M. (eds) Encyclopedia of Cancer. Springer, Berlin, Heidelberg. https://doi.org/10.1007/978-3-642-27841-9_6729-2
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DOI: https://doi.org/10.1007/978-3-642-27841-9_6729-2
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