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Ovarian Cancer pp 783-792 | Cite as

p53 Adenovirus as Gene Therapy for Ovarian Cancer

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Part of the Methods in Molecular Medicine™ book series (MIMM, volume 39)

Abstract

Ovarian cancer arises from the accumulation of mutations in multiple combinations of genes (1). The most extensively studied tumor suppressor gene in solid tumors is p53, a 53-kD nuclear phosphoprotein that binds DNA. The p53 gene product plays a role in normal cellular proliferation by regulating gene transcription, cell cycle control, and apoptosis (2). Mutations of p53 are the most common molecular genetic abnormality to be described in human cancer, and have been identified in malignancies of the breast, colon, lung, esophagus, head and neck, and hematopoietic system (3). Mutations of the p53 gene have been identified in 30 to 79% of epithelial ovarian cancers (4,5). Most of the mutations identified in p53 are distributed throughout the open reading frame as missense mutations. We have identified a missense mutation in the p53 gene in the 2774 ovarian cancer cell line that converts an arginine residue in the DNA binding region of the protein to a histidine residue (6). The mutation in codon 273 we found in 2774 cells is one of the six major hotspots identified for p53 missense mutations (7).

Keywords

Ovarian Cancer Epithelial Ovarian Cancer Ovarian Cancer Cell Ovarian Cancer Cell Line Peritoneal Washing 
These keywords were added by machine and not by the authors. This process is experimental and the keywords may be updated as the learning algorithm improves.

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Copyright information

© Humana Press Inc. 2000

Authors and Affiliations

  1. 1.Department of Cellular Biology and Anatomy, and Obstetrics and GynecologyLouisiana State University Medical Center- ShreveportShreveportLA
  2. 2.Department of Cellular Biology and Anatomy, and Division of Gynecologic Oncology, Department of Obstetrics and GynecologyLouisiana State University Medical Center-ShreveportShreveportLA
  3. 3.Department of Cellular Biology and AnatomyLouisiana State University Medical Center-ShreveportShreveport
  4. 4.Division of Gynecologic OncologyUniversity of Texas Medical BranchGalveston

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