Correction to: J Exp Clin Cancer Res 36, 147 (2017)
https://doi.org/10.1186/s13046-017-0621-2
Following publication of the original article [1], the authors identified some minor errors in Fig. 3 and Fig. S3, specifically:
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Figure 3e: incorrect images were used for colony formation assay images for CNE2 Vector, CNE2 ZNF671 and 5-8F ZNF671 (top left, top right, and bottom right)
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Fig. S3a: incorrect images were used for migration assay images for CNE2 ZNF671 at both 0 h and 48 h (left panel, bottom row)
Effects of ZNF671 overexpression on NPC cell viability and colony formation ability in vitro. a qPCR analysis of ZNF671 mRNA expression in CNE-2 and 5-8F cells stably overexpression ZNF671. b Western blotting analysis of ZNF671 expression in CNE-2 and 5-8F cells stably overexpression ZNF671. c-d The CCK-8 assay showed overexpression of ZNF671 reduced the viability of CNE2 (c) and 5-8F (d) cells. e The colony formation assay showed overexpression of ZNF671 suppressed colony-forming ability. All experiments were performed at least three times; data are mean ± SD. *P < 0.05, **P < 0.01 vs. control, Student’s t-test
The corrected figures are given here. The corrections do not have any effect on the final conclusions of the paper. The original article has been corrected.
Reference
Zhang J, Wen X, Liu N, et al. Epigenetic mediated zinc finger protein 671 downregulation promotes cell proliferation and tumorigenicity in nasopharyngeal carcinoma by inhibiting cell cycle arrest. J Exp Clin Cancer Res. 2017;36:147. https://doi.org/10.1186/s13046-017-0621-2.
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Additional file 3 Fig. S3.
ZNF671 has no effect on affect NPC migratory and invasive ability. (A) Migration ability was measured using a wound healing assay (200 ×) and (B) Transwell assay with Matrigel (200 ×) in CNE2 and SUNE1 cells with the vector or ZNF671 overexpression. Scale bar: 100 μm; data are mean ± SD. *P < 0.05, **P < 0.01 vs. control, Student’s t-test.
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Zhang, J., Wen, X., Liu, N. et al. Correction to: Epigenetic mediated zinc finger protein 671 downregulation promotes cell proliferation and tumorigenicity in nasopharyngeal carcinoma by inhibiting cell cycle arrest. J Exp Clin Cancer Res 40, 394 (2021). https://doi.org/10.1186/s13046-021-02205-0
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DOI: https://doi.org/10.1186/s13046-021-02205-0