Erratum to: BMC Genomics doi 10.1186/s12864-015-1526-0
Following the publication of our recent article in BMC Genomics [1] we wish to bring the following corrigendum to your attention. In the above paper, we wrote in the discussion on page 13: “These are V756F in XlnR (corresponding to V801 in XYR1) and A804V (based on our analysis; not A824V as stated by the authors) in XYR1”. Our analysis of the XYR1 sequence was based on the available Trichoderma reesei QM6a XYR1 sequences deposited in the NCBI database [Protein Accession Number XP_006966092.1 and EGR48040.1]. Recently, Derntl et al. [2] identified that the second intron in xyr1 is in fact not spliced thus giving rise to a protein that, while maintaining the reading frame, is 20 amino acids longer. Consequently, the position A824V given by Derntl et al. [2] is correct, and the numbering of amino acids in our paper after G319 has to be increased by 20.
A corrected version of our statement would read: "These are V756F in XlnR of A. niger which corresponds to V821 in T. reesei XYR1, as well as A824V in XYR1 of T. reesei [35, 42]".
We apologize for this mistake, but would like to stress that none of the results or conclusions in our paper are affected by this change.
References
Lichius A, Bidard F, Buchholz F, Le Crom S, Martin J, Schackwitz W, et al. Genome sequencing of the Trichoderma reesei QM9136 mutant identifies a truncation of the transcriptional regulator XYR1 as the cause for its cellulase-negative phenotype. BMC Genomics. 2015;16:326.
Derntl C, Gudynaite-Savitch L, Calixte S, White T, Mach RL, Mach-Aigner AR. Mutation of the xylanase regulator 1 causes a glucose blind hydrolase expressing phenotype in industrially used Trichoderma strains. Biotechnol Biofuels. 2013;6:62.
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Alexander Lichius and Frédérique Bidard contributed equally to this work.
The online version of the original article can be found under doi:10.1186/s12864-015-1526-0.
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Lichius, A., Bidard, F., Buchholz, F. et al. Erratum to: Genome sequencing of the Trichoderma reesei QM9136 mutant identifies a truncation of the transcriptional regulator XYR1 as the cause for its cellulase-negative phenotype. BMC Genomics 16, 725 (2015). https://doi.org/10.1186/s12864-015-1917-2
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DOI: https://doi.org/10.1186/s12864-015-1917-2