References
Battezzati A, Caumo A, Martino F, et al. Nonhepatic glucose production in humans. Am J Physiol Endocrinol Metab. 2004;286:E124–35.
Duraffourd C, De Vadder F, Goncalves D, et al. Mμ-opioid receptors and dietary protein stimulate a gut-brain neural circuitry limiting food intake. Cell. 2012;150:377–388.
Hayes MT, Foo J, Besic V, et al. Is intestinal gluconeogenesis a key factor in the early changes in glucose homeostasis following gastric bypass? Obes Surg. 2011;21:759–62.
Mithieux G. New data and concepts on glutamine and glucose metabolism in the gut. Curr Opin Clin Nutr Metab Care. 2001;4:267–71.
Mithieux G, Misery P, Magnan C, et al. Portal sensing of intestinal gluconeogenesis is a mechanistic link in the diminution of food intake induced by diet protein. Cell Metab. 2005;2:321–9.
Mithieux G, Andreelli F, Magnan C. Intestinal gluconeogenesis: key signal of central control of energy and glucose homeostasis. Curr Opin Clin Nutr Metab Care. 2009;12:419–23. Review.
Pillot B, Soty M, Gautier-Stein A, et al. Protein feeding promotes redistribution of endogenous glucose production to the kidney and potentiates its suppression by insulin. Endocrinology. 2009;150:616–24.
Troy S, Soty M, Ribeiro L, et al. Intestinal gluconeogenesis is a key factor for early metabolic changes after gastric bypass by not after gastric lap-band in mice. Cell Metab. 2008;8:201–11.
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Mithieux, G. Comment about intestinal gluconeogenesis after gastric bypass in human in relation with the paper by Hayes et al., Obes. Surg. 2011. OBES SURG 22, 1920–1922 (2012). https://doi.org/10.1007/s11695-012-0755-4
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DOI: https://doi.org/10.1007/s11695-012-0755-4