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Subclinical activation of lupus nephritis by recombinant human growth hormone

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Abstract.

The effect of growth hormone (GH) on subclinical disease activity in a 15-year-old boy with previously quiescent lupus nephritis and chronic renal failure is described. Institution of supraphysiological doses of GH resulted in a rise in erythrocyte sedimentation rate, decrease in serum complement, rise in anti-DNA antibody titers, and increase in T-cell activation markers, all of which improved following cessation of GH treatment.

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Authors and Affiliations

  1. Department of Pediatrics, National University of Singapore, Singapore, , , , , , SG

    Hui-Kim Yap, Kah-Yin Loke, Belinda Murugasu & Bee-Wah Lee

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  1. Hui-Kim Yap
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  2. Kah-Yin Loke
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  3. Belinda Murugasu
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  4. Bee-Wah Lee
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Additional information

Received April 3, 1997; received in revised form September 2, 1997; accepted September 3, 1997

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Yap, HK., Loke, KY., Murugasu, B. et al. Subclinical activation of lupus nephritis by recombinant human growth hormone. Pediatr Nephrol 12, 133–135 (1998). https://doi.org/10.1007/s004670050421

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  • DOI: https://doi.org/10.1007/s004670050421

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