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Psychoendocrine interrelationships in patients in a state of emotional stress during cerebral obesity

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Conclusions

  1. 1.

    Patients with HPRS and without it have different psychohormonal complexes of reaction to a state of stress, which corroborates the heterogeneity of pathogenetic mechanisms in increased body weight in a state of stress. In the presence of HPRS, increase in body weight in a state of ES basically occurs because of inadequate psychoemotional shifts with subsequent overeating. In the absence of such reactivity, increase in body weight in conditions of ES basically occurs through the neuroendocrine network (inhibited lipolysis).

  2. 2.

    Data obtained corroborate the importance of change in synthesis and reactivity of STH in pathogenetic mechanisms of obesity, which possibly bear a primary constitutionally governed character and is decompensated even more during existence under the influence of ES.

  3. 3.

    This investigation makes it necessary to formulate different approaches to treating patients with cerebral obesity depending on presence or absence of HPRS.

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Authors and Affiliations

  1. Department of Pathology of the Vegetative Nervous System, I. M. Sechenov First Moscow Medical Institute, USSR

    T. G. Voznesenskaya, A. D. Solov'eva & N. M. Fokina

Authors
  1. T. G. Voznesenskaya
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  2. A. D. Solov'eva
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  3. N. M. Fokina
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Additional information

Translated from Problemy Endokrinologii, Vol. 35, No. 1, pp. 3–7, January–February, 1989.

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Voznesenskaya, T.G., Solov'eva, A.D. & Fokina, N.M. Psychoendocrine interrelationships in patients in a state of emotional stress during cerebral obesity. Neurosci Behav Physiol 19, 408–412 (1989). https://doi.org/10.1007/BF01197873

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  • DOI: https://doi.org/10.1007/BF01197873

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