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Deficiency of toxin-binding protein activity in mutants of sugarcane clone H54-775 as it relates to disease resistance

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Abstract

Three mutants selected from a population of sugarcane clone H54-775 that had been irradiated with 3 kR γ-radiation all lacked toxin-binding protein activity. This activity previously had been shown to be essential for eye spot disease susceptibility and was demonstrated in the susceptible parent clone H54-775. In one mutant, the biochemical, immunochemical, and electrophoretic mobilities of the toxin-binding protein were all modified.

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The work reported here was supported in part by Grant GB 43192 from the National Science Foundation and Grant C145-2575 from the Cooperative States Research Service of the United States Department of Agriculture and the Hawaiian Sugar Planters' Association, Honolulu, Hawaii; Montana Agricultural Experiment Station, Paper No. 579, Journal Series, and Paper No. 370, Journal Series of the Experiment Station, HSPA.

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Strobel, G.A., Steiner, G.W. & Byther, R. Deficiency of toxin-binding protein activity in mutants of sugarcane clone H54-775 as it relates to disease resistance. Biochem Genet 13, 557–565 (1975). https://doi.org/10.1007/BF00484915

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  • DOI: https://doi.org/10.1007/BF00484915

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