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Organization and Expression of the hrp Gene Cluster in Pseudomonas Syringae Pv. Phaseolicola

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Vascular Wilt Diseases of Plants

Part of the book series: NATO ASI Series ((ASIH,volume 28))

Abstract

Infection of plants by pathogens generally leads to one or the other of two mutually exclusive outcomes, either the development of disease symptoms characteristic of the pathogen-host-environment combination (compatible interactions) or the triggering of the host resistance response (incompatible interaction). The resistance response is often typified by necrosis of host cells directly contacted by the pathogen or cells in their immediate vicinity at early stages of infection. Restriction of pathogen growth and development may result as a direct consequence of substances that are inhibitory to the pathogen, termed phytoalexins, as an indirect consequence of cell death per se, in the case of obligate pathogens, or via other less well-understood biochemical or cyto-developmental events that are characteristic of this response at the biochemical, cellular, or histological level. The triggering of hypersensitive death may be effected by pathogen- or plant-cell-derived necrosis-eliciting molecules whose nature, role and mechanisms of action are poorly understood at present.

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© 1989 Springer-Verlag Berlin Heidelberg

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Rahme, L., Mindrinos, M., Grimm, C., Frederick, R., Lindgren, P., Panopoulos, N. (1989). Organization and Expression of the hrp Gene Cluster in Pseudomonas Syringae Pv. Phaseolicola . In: Tjamos, E.C., Beckman, C.H. (eds) Vascular Wilt Diseases of Plants. NATO ASI Series, vol 28. Springer, Berlin, Heidelberg. https://doi.org/10.1007/978-3-642-73166-2_22

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  • DOI: https://doi.org/10.1007/978-3-642-73166-2_22

  • Publisher Name: Springer, Berlin, Heidelberg

  • Print ISBN: 978-3-642-73168-6

  • Online ISBN: 978-3-642-73166-2

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