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Potenzieller Podozytenschaden durch Bardoxolone in klinischen Studien

Genetische oder pharmakologische Aktivierung von Nrf2 erhöht bei chronisch nierenkranken Mäusen die Proteinurie und führt zu Podozytenschaden

Potential podocyte damage from bardoxolone in clinical trials

Genetic or pharmacological activation of Nrf2 increases proteinuria in mice with chronic kidney disease and leads to podocyte damage

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Literatur

  1. Baigent C, Lennon R (2018) Should we increase GFR with bardoxolone in Alport syndrome? J Am Soc Nephrol 29(2):357–359

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Authors and Affiliations

  1. Klinik für Nephrologie und Rheumatologie, Universitätsmedizin Göttingen, Robert-Koch-Str. 40, 37075, Göttingen, Deutschland

    Oliver Gross

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  1. Oliver Gross
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Correspondence to Oliver Gross.

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Interessenkonflikt

O. Gross war von 2018 bis 2020 Hauptprüfer für das einzige deutsche Prüfzentrum der CARDINAL Studie (Sponsor Reata Pharmaceuticals).

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Thorsten Feldkamp, Hann. Münden

Jens Lutz, Koblenz

Lutz T. Weber, Köln

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Gross, O. Potenzieller Podozytenschaden durch Bardoxolone in klinischen Studien. Nephrologe 17, 57–58 (2022). https://doi.org/10.1007/s11560-021-00548-5

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  • DOI: https://doi.org/10.1007/s11560-021-00548-5

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