Abstract
Traditionally, hyperlactatemia in critically ill patients and particularly those in shock was interpreted as a marker of secondary anaerobic metabolism due to inadequate oxygen supply inducing cellular distress [1]. Many arguments have since refuted this view [2]. With lactate metabolism being extensively described in classical biochemistry manuals, this chapter will focus only on those aspects as they relate to critically ill patients. Distinction between lactic acidosis, metabolic acidosis with hyperlactatemia, and isolated hyperlactatemia will also be addressed.
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Levy, B. (2006). Lactic Acidosis and Hyperlactatemia. In: Vincent, JL. (eds) Intensive Care Medicine. Springer, New York, NY. https://doi.org/10.1007/0-387-35096-9_9
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DOI: https://doi.org/10.1007/0-387-35096-9_9
Publisher Name: Springer, New York, NY
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