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Abstract

Episodes of symptomatic circulatory collapse occur in 25% of dialysis sessions. They not only compromise the patients well-being, but also interfere with the attempts of the dialysis team to reach and maintain dry weight. Basically there is nothing mysterious about dialysis hypotension (DH). Depending on the state of overhydration, the removal of 2–4 L of extracellular fluid is accompanied by a 10–30% decrease in blood volume in a few hours. It can be imagined that circulatory adaptation to this unphysiological change may occasionally fall short of complete compensation. It is logical therefore, that the occurrence of DH will be greatly enhanced by two events: 1. Insufficient dietary salt restriction causing excessive fluid gain and 2. Short dialysis.

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Bibliography

  • Akahüseyin E, Nette RW, Vincent HH et al. A simulation study on the intercomparimental fluid shifts during hemodialysis. ASAIO Journal. 2000; 46: 81–94.

    Article  Google Scholar 

  • Barnas MGW, Boer WH, Schelven LJ et al. Heart rate variability during dialysis hypotension: confirmation on Bezold—Jarish-like reflex. Kidney Int. 1999; 55: 2602.

    Google Scholar 

  • Daul AE, Wang XL, Michel MC. Arterial hypotension in dialysis patients. Kidney Int. 1987; 32: 728–35.

    Article  CAS  PubMed  Google Scholar 

  • Daugirdas JT. Dialysis hypotension: a hemodynamic analysis. Kidney Int. 1991; 39: 233–46.

    Article  CAS  PubMed  Google Scholar 

  • Esforzado N, Cases A, Bono M et al. Vasoactive hormones in uremic patients with chronic hypotension. Nephrol Dial Transplant. 1997; 12: 321–4.

    Article  Google Scholar 

  • Katzarski KS, Nisei] J, Randmaa I et al. Critical evaluation of ultrasound measurement of inferior vena cava diameter in assessing dry weight in normotensive and hypertensive hemodialysis patients. Am J Kidney Dis. 1997; 30: 459–65.

    Article  CAS  PubMed  Google Scholar 

  • Kooman JP, Wijnen JAG, Draayer Petal. Compliance and reactivity of the peripheral venous system in chronic intermittent haemodialysis. Kidney Int. 1992; 41: 1041–8.

    Article  CAS  PubMed  Google Scholar 

  • Koomans HA, Geers AB, Dorhout Mees EJ. Plasma volume recovery after ultrafiltration in patients with chronic renal failure. Kidney Int. 1984; 26: 848–54.

    Article  CAS  PubMed  Google Scholar 

  • Moore TJ, Lazarus JM, Hakim RM. Reduced angiotensin receptors and pressor responses in hypotensive hemodialysis patients. Kidney Int. 1989; 36: 696–701.

    Article  CAS  PubMed  Google Scholar 

  • Ruffmann K, Mandelbauam A, Bommer J et al. Doppler echocardiographic findings in dialysis patients. Nephrol Dial Transplant. 1990; 5: 426–31.

    Article  CAS  PubMed  Google Scholar 

  • van der Sande FM, Cheriex EC, van Kuik WHM et al. Effect of dialysate calcium concentrations on intradialytic blood pressure course in cardiac-compromised patients. Am J Kidney Dis. 1998; 32: 125–31.

    Article  PubMed  Google Scholar 

  • Santoro A, Manchini E, Spongano M et al. A haemodynamic study of hypotension during haemodialysis. Nephrol Dial Transplant. 1990;(Suppl. 1 ): 147–53.

    Google Scholar 

  • Verresen L, Fink E, Lemke H et al. Bradykinin is a mediator of anaphylactoid reactions during hemodiaysis with AN 69 membranes. Kidney Int. 1994; 45: 1497–503.

    Article  CAS  PubMed  Google Scholar 

  • Wizemann V, Soetanto R, Thormann J et al. Effects of acetate on left ventricular function in hemodialysis patients. Nephron. 1993; 64: 101–05.

    Article  CAS  PubMed  Google Scholar 

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© 2000 Springer Science+Business Media Dordrecht

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Mees, E.J.D. (2000). Dialysis hypotension. In: Cardiovascular Aspects of Dialysis Treatment. Springer, Dordrecht. https://doi.org/10.1007/978-94-017-0973-6_6

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  • DOI: https://doi.org/10.1007/978-94-017-0973-6_6

  • Publisher Name: Springer, Dordrecht

  • Print ISBN: 978-90-481-5442-5

  • Online ISBN: 978-94-017-0973-6

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