Abstract
Most schizophrenia research is currently conducted within the diathesis-stress framework wherein some portion of variation in phenotypic schizophrenia is assumed to be due to environmental factors and some portion due to genetic factors. The specific components of this model, however, have not been well-characterized. Although genetic research offers the most solid evidence concerning etiology, the mode of transmission is unclear (Matthysse & Kidd, 1976) and the biochemical or psychological features which constitute vulnerability (diathesis) have not been delimited. On the other hand, research concerning environmental determinants has not contributed substantially to our understanding of the disease (Neale and Oltmanns, 1980). It follows that it remains unclear how genetic and environmental factors interact. Is the genotype represented in different phenotypes? If so, how does the genotype interact with stressors? Schizophrenia spectrum illnesses are a potentially fruitful way of studying the interplay between constitutional vulnerability and stressors. Spectrum illnesses may represent genotypic schizophrenia. By comparing persons who are genotypically similar and phenotypic ally different, it is possible to elucidate environmental factors important in shaping outcome. The present study is concerned with one possible spectrum illness, criminal or antisocial behavior.
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© 1988 Martinus Nijhoff Publishers, Dordrecht
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Silverton, L. (1988). Crime and the Schizophrenia Spectrum: A Study of Three Danish Cohorts. In: Moffitt, T.E., Mednick, S.A. (eds) Biological Contributions to Crime Causation. NATO ASI Series, vol 40. Springer, Dordrecht. https://doi.org/10.1007/978-94-009-2768-1_11
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