Zusammenfassung
Eine wesentliche Eigentümlichkeit des Herzmuskels ist die Fähigkeit der Anpassung seiner Tätigkeit an die Erfordernisse des Organismus. Diese Anpassungsfähigkeit macht das Leben erst möglich, da sie dem Herzen die Vergrößerung des Schlag- und Minutenvolumens gestattet, wodurch es größeren Beanspruchungen während körperlicher Anstrengung, Verdauung, Schwangerschaft und zahlreicher anderer Situationen nachzukommen vermag. Die vom Herzen geförderte Blutmenge kann von 5 bis 7 Litern in der Minute bei Ruhe ohne weiteres auf 25 bis 30 Liter bei starker Anstrengung ansteigen. Der Herzmuskel nützt bei Ruhe nicht alle seiner Möglichkeiten aus; er verfügt über eine Reservekraft, welche immer dann zur Anwendung kommt, wenn die Füllung des Herzens zunimmt. Innerhalb gewisser Grenzen gilt für das gesunde Herz das Starlingsche Gesetz. Eine stärkere Dehnung der Herzmuskelfasern infolge vermehrter Herzfüllung löst eine kräftigere Kontraktion aus.
Access this chapter
Tax calculation will be finalised at checkout
Purchases are for personal use only
Preview
Unable to display preview. Download preview PDF.
Schrifttum
Alexandresco-Dersca, C., et Focsa, P. “Trois cas de syndrome de Bernheim.” Presse méd., 39, 1437, 1931.
Anrep, G. V., and Segall, H. N. “The Central and Reflex Regulation of the Heart Rate.” J. Physiol., 61, 215, 1926.
Bainbridge, F. A. “The Influence of Venous Filling upon the Rate of the Heart.” J. Physiol. 50, 65, 1915.
Barath, E., und Elias, H Klinische Beiträge zum Verhalten des onkotischen (kolloidosmotischen) Druckes; der onkotische Druck im Serum des arteriellen und venösen Blutes bei Kreislaufstörungen. Ztschr. f. klin. Med., 114, 708, 1930.
Bernheim. “De l’asystolie veineuse dans l’hypertrophie du coeur gauche par sténose concomitante du ventricule droit.” Rev. de méd., 30, 785, 1910.
Blake, W. D. and others, Effect of increased renal venous pressure on renal function, Am. J. Physiol. 157, 1, 1949.
Bolton, C. “The Absorption of Fluid in Cardiac Dropsy.” Heart, 11, 343, 1924.
Bornstein, J. and Trewhella, P., Adrenocorticotropic activity of blood-plasma extracts, Lancet, 2, 678, 1950.
Borst, J. G. G., The maintenance of an adequate cardiac output by the regulation of the urinary excretion of water and sodium chloride; an essential factor in the genesis of oedema, Acta med. scand. Suppl. 207, 1948.
Boyer, N. H., and White, P. D. “Right-upper-quadrant Pain on Effort: An Early Symptom of Failure of the Right Ventricle.” New England J. Med., 226, 217, 1942.
Bradley, S. E. and Blake, W. D., Pathogenesis of renal dysfunction during con-gestive heart failure, Am. J. Med., 6, 470, 1949.
Brenner, O. “Pathology of the Vessels of the Pulmonary Circulation” Arch. Int. Med., 56, 211, 1935.
Burch, G. E. and Ray, C. T., A consideration of the mechanism of congestive heart failure, Am. Heart J., 41, 918, 1951.
Cohn, A. E., and Steele, J. M. “Unexplained Fever in Heart Failure.” J. Clin. Investigation, 13, 853, 1934.
Cossio, P., and Bereonsky, I. “The Cyanosis in Mitral Stenosis.” Am. Heart. J., 17, 1, 1939.
Curschmann, H. Über klimakterisches Odem. Med. Klin., 29, 1270, 1933.
Davies, C. E., and Kilpatrick J. A., Renal circulation in “low output” and “high output” heart failure, Clin. Science, 10, 53, 1951.
Day, T. D., and Armstrong, T. C. “Fibrosis of the Liver in Heart Failure.” J. Path. & Bact., 50, 221, 1940.
Dock, W., Congestive heart failure, J. A. M. A., 140, 1135, 1949.
Drury, A. N., and Jones, N. W. “Observations on the Rate at which Oedema forms when the Veins of the Human Limb are Congested.” Heart, 14, 55, 1927.
East, T. and Bain, C., Right ventricular stenosis (Bernheim’s syndrome), Brit. Heart J., 11, 145, 1949.
East, T., “Pulmonary Hypertension.” Brit. Heart J., 2, 189, 1940.
Editorixal, Hydrothorax in congestive heart failure, Am. J. Roentgen., 60, 419, 1948.
Elias, H., und Feller, A. Stauungstypen bei Kreislaufstörungen. Mit besonderer Berücksichtigung der exsudativen Perikarditis. Wien, J. Springer, 1926.
Eppinger, H. in Kraus und Brugsch, Spezielle Pathologie und Therapie, Berlin, Urban und Schwarzenberg, 1920.
Eufinger, H., und Spiegler R. Der Einfluß des mensuellen Zyklus auf den Wasserstoffwechsel. Arch. f. Gynäk., 135, 223, 1928.
Evans, J. M. and others, Altered liver function of chronic congestive heart failure, Am. J. Med., 13, 704, 1952.
Eyster, J. A. E. “Cardiac Dilatation and Hypertrophy.” Tr. A. Am. Physicians, 42, 15, 1927.
Fishberg, A. M. “Jaundice in Myocardial Insufficiency.” J. A. M. A. 80, 1516, 1923
Fishberg, A. M. “Heart Failure.” Philadelphia, Lea & Febiger, 1937.
Fitzgerald Peel, A. A., Dissecting aneurysm of the inter ventricular septum, Brit. Heart J., 10, 239, 1948.
Frank, O. Zur Dynamik des Herzmuskels. Ztschr. f. Biol., 32, 370, 1895.
Grassmann, W., und Herzog, F. Die Wirkung von Digitalis (Strophanthin) auf das Minuten-und Schlagvolumen des Herzkranken. Arch. f. exper. Path. u. Phar-makol, 163, 97, 1931.
Hanenson, I. B. et al., Pathogenesis and treatment of congestive heart failure, M. Clin. North America, p. 643, May 1953.
Henderson, Y., and Prince, A. L. “The Relative Systolic Discharge of the Right and Left Ventricles and their Bearing on Pulmonary Congestion and Depletion.” Heart, 5, 217, 1913–14.
Hickam, J. B. and Cargill, W. H., Effect of exercise on cardiac output and pulmonary artercal pressure in normal persons and in patients with cardiovascular diseases and pulmonray emphysema, J. Clin. Invest. 27, 10, 1948.
Jouve, A., and Vague, J., La circulation de retour, Paris, Masson, 1940.
Kagan, B. M. “The Serum Proteins in Diseases of the Heart and Kidneys.” Am. J. Clin. Path., 14, 327, 1944.
Katzin, H. M., Waller, J. V. and Blumgart, H. L. “Cardiac Cirrhosis of the Liver.” Arch. Int. Med., 64, 457, 1939.
Keefer, C. S., and Resnik, W. H. “Jaundice following Pulmonary infarction in. Patients with Myocardial Insufficiency.” J. Clin. Investigation, 2, 375, 389, 1926.
Kinsey, D., and White, P. D. “Fever in Congestive Heart Failure.” Arch. Int. Med., 65, 163, 1940.
Krogh, A., Landis, E. M., and Turner, A. H. “The Movement of Fluid through the Human Capillary Wall in Relation to Venous Pressure and to the Colloid Osmotic -Pressure of the Blood.” J. Clin. Investigation, 11, 63, 1932.
Kugel, M. A., and Lichtman, S. S. “Factors causing Clinical Jaundice in Heart Disease.” Arch. Int. Med., 52, 16, 1933.
Landis, E. M. et al., Central venous pressure in relation to cardiac “competence”, blood volume and exercise, J. Clin. Invest., 25, 237, 1946.
Lundsgaard, C., and Van Slyke, D. D. “Cyanosis.” Medicine, 2, 1, 1923.
McMichael, J. The output of the heart in congestive failure, Quart. J. Med., 7, 331, 1938.
Meakins, J. C. “Distribution of Jaundice in Circulatory Failure” J. Clin. Investigation, 4, 135, 1927.
Mellinkoff, S. M. and Tumulty, P. A., Hepatic hypoglycemia in heart failure, New Eng. J. Med., 247, 745, 1952.
Merrill, A. J.. Edema and decreased renal blood flow in patients with chronic congestive heart failure; Evidence of “forward failure” as the primary cause of edema, J. Clin. Invest., 25, 389, 1946.
Miller, G. E., Water and electrolyte metabolism in congestive heart failure, Circulation, 4, 270, 1951.
Mokotoff, R., Ross, G. and Leiter, L., Renal plasma flow and sodium reabsorption and excretion in congestive heart failure, J. Clin. Invest., 27, 1, 1948.
de Navasquez, S., Forbes, J. R., and Rolling, H. E. “Right Ventricular Hypertrophy of Unknow Origin: So-called Pulmonary Hypertension.” Brit. Heart, J., 2, 177, 1940.
Page, I. H. “Ipsolateral Oedema and Contralateral Jaundice Associated with Hemiplegia and Cardiac Decompensation.” Am. J. M. Sc., 177, 273, 1929.
Peters, J. P., The problem of cardiac edema, Am. J. Med., 12, 66, 1952.
Rich, A. R., and Resnik, W. H. “On the Mechanism of the Jaundice following Pulmonary Infarction in Patients with Heart Failure” Bull. Johns Hopkins Hosp., 38, 75, 1926.
Raab, W., Hormonal and Neurogenic Cardiovascular disorders, Baltimore, Williams & Wilkins, 1953.
Schalm, L. and Hoogenboom, W. A. H., Blood blirubin in congestive heart failure, Am. Heart J., 44, 571, 1952.
Schön, R. Experimentelle Untersuchungen über Meteorismus. Deutsches Arch. f. klin. Med., 148, 86, 1925.
Schroeder, H. A., Studies on congestive circulatory failure, Circulation 1, 1481, 1950.
Selzer, A., Chronic cyanosis, Am. J. Med., 10, 334, 1951.
Seymour, W. B., and others, Cardiac output, blood and interstitial fluid volumes, total circulating serum protein and kidney function during cardiac failure and after improvement, J. Clin. Invest., 21, 229, 1942.
Sherlock, S., The liver in heart failure, Brit. Heart J., 13, 273, 1951.
Sinclair-Smith, B. and others, The renal mechanism of electrolyte excretion and the metabolic balances of electrolytes and nitrogen in congertive cardiac failure; the effects of exercise, rest and aminophyllin. Bull. Johns Hopkins Hosp., 84, 369, 1949.
Singer, B. and Wener, J., Excretion of sodium-retaining substances in patients with congestive heart failure, Am. Heart J., 45, 795, 1953.
Smirk, F. H. “Observations on the Causes of Oedema in Congestive Heart Failure.” Clin. Sc., 2, 317, 1936.
Sodeman, W. A., and Burch, G. E. “The Precipitating Causes of Congestive Heart Failure.” Am. Heart J., 15, 22, 1938.
Starling, E. H. The Arris and Gale lectures on some points in the pathology of heart disease, Lana, et, 562, 569, 723, 1897.
Starr, I. and Rawson, A. J., Role of the “Static Blood Pressure” in abnormal increments of venous pressure, especially in heart failure, Am. J. Med., Scienc. 199, 27 and 40, 1940.
Starr, I., Jeffers, W. A. and Meade, R. H. Jr., The absence of conspicuous increments of venous pressure after severe damage tote right ventricle of the dog, Am. Heart J., 26, 291, 1943.
Stewart, H. J., and Moore, N. S. “The Number of Formed Elements in the Urinary Sediment of Patients suffering from Heart Disease, with Particular Reference to the State of Heart Failure.” J. Clin. Investigation, 9, 409, 1931.
Sweeney, J. S. “Menstrual Oedema.” J. A. M. A., 103, 234, 1934.
Sylla, A. Lungenstauung und Stauungslunge. Ergebn. d. inn. Med. u. Kindern., 49, 122, 1935.
Thorn, G. W. and others, a study of mechanism of edema associated with menstruation, Endocrinology, 22, 155, 1938.
Thorn, G. W., Nelson, K. R., and Thorn, D. W. “A Study of the Mechanism of Oedema Associated with Menstruation.” Endocrinology, 22, 155, 1938.
Warren, J. V. and Stead, E. A. Jr., Fluid dynamics in chronic congestive heart failure, Arch., int. Med., 73, 138, 1944.
Wollheim, E. Die zirkulierende Blutmenge und ihre Bedeutung für die Kompensation und Dekompensation des Kreislaufes. Ztschr. f. klin. Med., 116, 269, 1931.
Zdansky, E. Beiträge zur Kenntnis der kardialen Lungenstauung auf Grund röntgenologischer, klinischer und anatomischer Untersuchungen. Wien, Arch. f. inn. Med., 18, 461, 1929.
Zdansky, E., Röntgendiagnostik des Herzens und der großen Gefäße. Springer-Verlag, Wien 1949, 2. Aufl.
Zdansky, E., Die Funktion des Herzens im Roentgenbilde, Fortschr. Geb. d. Roentgenstr. 76, 295, 1952.
Zdansky, E., Verh. d. Ges. f. Kreislfschg. 1951.
Author information
Authors and Affiliations
Rights and permissions
Copyright information
© 1955 Springer-Verlag
About this chapter
Cite this chapter
Scherf, D., Boyd, L.J. (1955). Kompensation und Dekompensation. In: Klinik und Therapie der Herzkrankheiten und der Gefäßerkrankungen. Springer, Vienna. https://doi.org/10.1007/978-3-7091-7854-6_5
Download citation
DOI: https://doi.org/10.1007/978-3-7091-7854-6_5
Publisher Name: Springer, Vienna
Print ISBN: 978-3-7091-7855-3
Online ISBN: 978-3-7091-7854-6
eBook Packages: Springer Book Archive