Abstract
Several pathogenetic mechanisms have been proposed to explain the peculiar histological features of brain ischemia, i.e., the selective neuronal death and the so-called maturation phenomenon [17] — the extension of the lesions during the reperfusion.
On leave from Department of Neurology, University of Debrecen, Hungary; stipendary of Alexander von Humboldt Foundation, Bonn, FRG.
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Németh, G. et al. (1989). Immunocytochemical Studies on Some Brain Regions in a Rat Model of Incomplete Cerebral Ischemia. Relationship to Behavior and Aging. In: Hartmann, A., Kuschinsky, W. (eds) Cerebral Ischemia and Calcium. Springer, Berlin, Heidelberg. https://doi.org/10.1007/978-3-642-85863-5_11
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DOI: https://doi.org/10.1007/978-3-642-85863-5_11
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