Abstract
The pathophysiology of traumatic shock, like that of severe intraoperative stress, is closely related to the development of a systemic inflammatory response syndrome (SIRS), which can lead to a subsequent organ dysfunction syndrome, which in turn can finally result in a multiorgan dysfunction syndrome (MODS) [1].
Access this chapter
Tax calculation will be finalised at checkout
Purchases are for personal use only
References
Bone RC, Balk RA, Cerra FB, Dellinger RP, Fein AM, Knaus WA, Schein RMH, Sibbald WJ (1992) Definitions for sepsis and organ failure and guidelines for the use of innovative therapies in sepsis. Chest 101:1644–1655.
Redl H, Schlag G (1991) Pathophysiology of multiorgan failure — inflammatory factors and mediators. Clin Intensive Care 2:16–27.
Schlag R, Redl H, Dinges HP, Davies J (1991) Sources of endotoxin in the posttraumatic setting. In: Levin J, Sturk A, ten Cate J (eds) Bacterial endotoxins: cytokine mediators and new therapies for sepsis. Wiley/Liss, New York, pp 121–134.
Author information
Authors and Affiliations
Editor information
Editors and Affiliations
Rights and permissions
Copyright information
© 1993 Springer-Verlag Berlin Heidelberg
About this chapter
Cite this chapter
Schlag, G., Redl, H. (1993). Introduction: “Organ in Shock”, “Early Organ Failure”, “Late Organ Failure”. In: Schlag, G., Redl, H. (eds) Pathophysiology of Shock, Sepsis, and Organ Failure. Springer, Berlin, Heidelberg. https://doi.org/10.1007/978-3-642-76736-4_1
Download citation
DOI: https://doi.org/10.1007/978-3-642-76736-4_1
Publisher Name: Springer, Berlin, Heidelberg
Print ISBN: 978-3-642-76738-8
Online ISBN: 978-3-642-76736-4
eBook Packages: Springer Book Archive