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The Use of Cardiovascular Drugs Following Thrombolysis in Humans

  • Conference paper
Limitation of Infarct Size

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Abstract

Acute myocardial infarction is the consequence of complete occlusion of a major coronary artery or a primary branch thereof, with resultant necrosis in the territory downstream [1]. Acute occlusion is almost always associated with the development of fresh thrombus proximate to an area of atherosclerosis. It appears likely that thrombosis is a consequence of ulceration of the surface of the thrombus usually anchored firmly in clefts or spaces within the atherosclerotic lesion. Extension into the lumen of the vessel in the presence of relatively severe atherosclerosis will cause complete occlusion; less severe degrees of atherosclerosis can also be associated with development of thrombus of a sufficient size to cause complete coronary artery occlusion.

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Authors
  1. H. J. C. Swan
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  2. A. S. Lew
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Editor information

Editors and Affiliations

  1. Dept. of Cardiology, University Hospital Rudolf Virchow, Spandauer Damm 130, D-1000, Berlin 19, Germany

    Horst Schmutzler , Wolfgang Rutsch  & Frank Christopher Dougherty ,  & 

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© 1989 Springer-Verlag Berlin Heidelberg

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Swan, H.J.C., Lew, A.S. (1989). The Use of Cardiovascular Drugs Following Thrombolysis in Humans. In: Schmutzler, H., Rutsch, W., Dougherty, F.C. (eds) Limitation of Infarct Size. Springer, Berlin, Heidelberg. https://doi.org/10.1007/978-3-642-73585-1_22

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  • DOI: https://doi.org/10.1007/978-3-642-73585-1_22

  • Publisher Name: Springer, Berlin, Heidelberg

  • Print ISBN: 978-3-540-19148-3

  • Online ISBN: 978-3-642-73585-1

  • eBook Packages: Springer Book Archive

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