Abstract
It is well known that hyperventilation decreases cerebral blood flow (CBF) by increasing cerebrovascular resistance. Positive pressure hyperventilation produces not only hypocapnia but also a rise in mean intrathoracic pressure, a decrease in venous return, with subsequent increase in central venous pressure (GVP), and a decrease in cardiac output (1).
This is a preview of subscription content, log in via an institution to check access.
Access this chapter
Tax calculation will be finalised at checkout
Purchases are for personal use only
References
Morgan, B. C., Martin, W. E., Hornheim, T. F., Crawford, E. W., and Guntheroth, W. G.: “Hemodynamic effects of intermittent positive pressure respiration.” Anaesthesiology 27:584 (1966).
Raichle, M. E., Posner, J. B., and Plum, F.: “Cerebral blood flow during and after hyperventilation.” Arch. Neurol. 23:394 (1970).
Rapela, C. E., and Green, H. D.: “Autoregula-tion of cerebral blood flow.” Circ. Res. 15:205 (1964).
Editor information
Editors and Affiliations
Rights and permissions
Copyright information
© 1975 Springer-Verlag New York Inc.
About this chapter
Cite this chapter
Schutz, H., Stoyka, W.W. (1975). Cerebrovascular Response to Hypocapnia in Normal and Brain-Injured Dogs. In: Langfitt, T.W., McHenry, L.C., Reivich, M., Wollman, H. (eds) Cerebral Circulation and Metabolism. Springer, Berlin, Heidelberg. https://doi.org/10.1007/978-3-642-65814-3_32
Download citation
DOI: https://doi.org/10.1007/978-3-642-65814-3_32
Publisher Name: Springer, Berlin, Heidelberg
Print ISBN: 978-3-642-65816-7
Online ISBN: 978-3-642-65814-3
eBook Packages: Springer Book Archive