Abstract
George Maton in 1814 realized that there was a mild illness characterized by rash, adenopathy and no fever that was distinct from scarlatina. This new disease was named rubella by Henry Veale in 1866. In 1942 an Australian ophthalmologist, Norman Gregg, noticed that German measles (rubella) infection in the first trimester of pregnancy caused serious birth defects in the offspring. The rubella virus was isolated in tissue culture in 1962 at two different laboratories. After the severe rubella epidemic in 1964, it was recognized that congenital rubella syndrome (CRS) included not only cardiovascular lesions, cataracts, deafness, mental retardation, central nervous system abnormalities and generalized growth retardation, but also bone lesions, hepatitis, meningoencephalitis, progressive rubella panencephalitis and eventual diabetes mellitus. In 1969, attenuated rubella vaccines became available for use in the United States (Cooper, 1985).
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Hethcote, H.W. (1989). Rubella. In: Levin, S.A., Hallam, T.G., Gross, L.J. (eds) Applied Mathematical Ecology. Biomathematics, vol 18. Springer, Berlin, Heidelberg. https://doi.org/10.1007/978-3-642-61317-3_9
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DOI: https://doi.org/10.1007/978-3-642-61317-3_9
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