Disc Hemorrhage as a Risk Factor for Progression of Normal-Tension Glaucoma
The pathogenesis of normal-tension glaucoma (NTG) is believed to be multifactorial. A variety of clinical factors have been implicated in the progression of optic nerve change in NTG. Intraocular pressure (IOP) plays a substantial role even in NTG eyes, which is supported by clinical evidence [1–6]. Factors other than IOP also seem to be involved in the development of glaucomatous optic neuropathy in at least a considerable number of NTG eyes. Included as the positive evidence for such factors are: a higher incidence of disc hemorrhage (DH) [7–12], more pronounced peripapillary chorioretinal atrophy , a higher incidence of retinal occlusive vascular diseases [9, 14], the development of glaucoma-like cupping after anterior ischemic optic neuropathy caused by giant cell arteritis [15–17], the frequently observed coexistence of several immunocompromised conditions [18, 19], the increased resistance index in orbital vessels , and an alteration in the diurnal curve of systemic blood pressure [21, 22].
KeywordsGiant Cell Arteritis Visual Field Loss Finger Blood Flow Anterior Ischemic Optic Neuropathy Disc Hemorrhage
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