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Disc Hemorrhage as a Risk Factor for Progression of Normal-Tension Glaucoma

  • Y. Kitazawa
  • T. Yamamoto
  • K. Ishida
Conference paper

Abstract

The pathogenesis of normal-tension glaucoma (NTG) is believed to be multifactorial. A variety of clinical factors have been implicated in the progression of optic nerve change in NTG. Intraocular pressure (IOP) plays a substantial role even in NTG eyes, which is supported by clinical evidence [1–6]. Factors other than IOP also seem to be involved in the development of glaucomatous optic neuropathy in at least a considerable number of NTG eyes. Included as the positive evidence for such factors are: a higher incidence of disc hemorrhage (DH) [7–12], more pronounced peripapillary chorioretinal atrophy [13], a higher incidence of retinal occlusive vascular diseases [9, 14], the development of glaucoma-like cupping after anterior ischemic optic neuropathy caused by giant cell arteritis [15–17], the frequently observed coexistence of several immunocompromised conditions [18, 19], the increased resistance index in orbital vessels [20], and an alteration in the diurnal curve of systemic blood pressure [21, 22].

Keywords

Giant Cell Arteritis Visual Field Loss Finger Blood Flow Anterior Ischemic Optic Neuropathy Disc Hemorrhage 
These keywords were added by machine and not by the authors. This process is experimental and the keywords may be updated as the learning algorithm improves.

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Copyright information

© Springer-Verlag Berlin Heidelberg 1999

Authors and Affiliations

  • Y. Kitazawa
  • T. Yamamoto
  • K. Ishida

There are no affiliations available

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