Zusammenfassung
Um die Pathogenese der mit vaskulärer Hypertrophie assoziierten genetischen Hypertonie zu klären, wurde in der vorliegenden Studie die Inkorporation von Aminosäure in das vaskuläre Protein altersstandardisierter normotensiver Ratten und hypertensiver Ratten, von denen einige eine blutdrucksenkende Behandlung hauptsächlich mit Clonidin erhielten, gemessen.
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© 1983 Dr. Dietrich Steinkopff Verlag, GmbH & Co. KG, Darmstadt
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Nakada, T., Lovenberg, W. (1983). Clonidin bei vaskulärer Hypertrophie. In: Hayduk, K., Bock, K.D. (eds) Zentrale Blutdruckregulation durch α 2-Rezeptorenstimulation. Steinkopff. https://doi.org/10.1007/978-3-642-48440-7_15
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DOI: https://doi.org/10.1007/978-3-642-48440-7_15
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