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Lipids as Effectors and Mediators in Growth Control of Ascites Tumor Cells

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Abstract

The growth requirement of some cell types for preformed cholesterol, normally supplied by the serum, has been established1–5. Cholesterol, which is a major component in the plasma membrane of eukaryotes, modulates bilayer fluidity6, and affects the activity of membrane-bound enzymes7. It can also modulate e.g. the characteristics of the beta-adrenergic receptor8.

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Haeffner, E.W., Strosznajder, J.B., Hoffmann, C.J.K. (1988). Lipids as Effectors and Mediators in Growth Control of Ascites Tumor Cells. In: Feo, F., Pani, P., Columbano, A., Garcea, R. (eds) Chemical Carcinogenesis. Springer, Boston, MA. https://doi.org/10.1007/978-1-4757-9640-7_53

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  • DOI: https://doi.org/10.1007/978-1-4757-9640-7_53

  • Publisher Name: Springer, Boston, MA

  • Print ISBN: 978-1-4757-9642-1

  • Online ISBN: 978-1-4757-9640-7

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