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Sodium Appetite Induced by Sodium Depletion is Suppressed by Intracerebroventricular Captopril

  • Mark L. Weiss
Part of the NATO ASI Series book series (NSSA, volume 105)

Abstract

We have previously shown1 that systemic captopril (CAP) has effects that implicate angiotensin II in the expression of sodium appetite induced by sodium depletion in the rat. In addition, that report suggested that it is angiotensin II (Ang II) in the brain that was important.

Keywords

Drinking Test Sodium Depletion Chemical Injection Drinking Response NaC1 Intake 
These keywords were added by machine and not by the authors. This process is experimental and the keywords may be updated as the learning algorithm improves.

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References

  1. 1.
    K. Moe, M.L. Weiss and A.N. Epstein, Sodium appetite during captopril blockade of endogenous angiotensin II formation, Am. J. Physiol., in press (1984).Google Scholar
  2. 2.
    J.T. Fitzsimons, A.N. Epstein and A.K. Johnson, Brain Res. 153: 319–331 (1978).PubMedCrossRefGoogle Scholar
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    W.A. Pettinger, M. Marchelle and L. Augusto, Am. J. Physiol. 221: 1071–1074 (1971).PubMedGoogle Scholar
  4. 4.
    M.L. Weiss, K. Moe and A.N. Epstein, Central blockade of angiotensin II’s actions suppresses the expression of sodium appetite induced by sodium depletion. Am J. Physiol., (1985).Google Scholar
  5. 5.
    J. Buggy and J. Jonklaas, Sodium decreased by central angiotensin blockade, Physiol. Behav., 32: (5)-737-742.Google Scholar

Copyright information

© Springer Science+Business Media New York 1986

Authors and Affiliations

  • Mark L. Weiss
    • 1
  1. 1.Department of Biology and Institute of Neurological SciencesUniversity of PennsylvaniaPhiladelphiaUSA

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