Abstract
Amyloidosis is an occasional sequela to long standing inflammation. The origin of the disorder is multifactorial; the etiology and the duration of inflammation, structure of the serum amyloid A (SAA) precursor, and genetic background of the host are all thought to contribute to amyloid deposition (1-4).
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Sipe, J.D., Rokita, H., Shirahama, T., Cohen, A.S. (1988). Deposition of Amyloid a Fibrils in Spleen is Accompanied by Decreased Hepatic and Splenic and Increased Macrophage Serum Amyloid a Expression. In: Isobe, T., Araki, S., Uchino, F., Kito, S., Tsubura, E. (eds) Amyloid and Amyloidosis. Springer, Boston, MA. https://doi.org/10.1007/978-1-4757-0298-9_14
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DOI: https://doi.org/10.1007/978-1-4757-0298-9_14
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