Studies of Dr. Hodson indicated that some type of functional obstruction causes segmental fibrosis. Could the Panel help clarify the roles played by reflux and by obstruction? Also, are there any differences in the vessels of the atrophic-fibrotic pole as compared with the rest of the kidney that would explain the hypertension found in these patients?
KeywordsVascular Change Membranous Nephropathy Raji Cell Panel Discussion Acute Pyelonephritis
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